Hypoxia induces netrin-1 and Unc5b in atherosclerotic plaques: mechanism for macrophage retention and survival
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Netrin-1 as a novel therapeutic target in cardiovascular disease: to activate or inhibit?1,25(OH)2D3 suppresses COX-2 up-regulation and thromboxane production in placental trophoblast cells in response to hypoxic stimulation.Netrins and their roles in placental angiogenesisAn uncoordinated-5 homolog B receptor monoclonal antibody regulates A375 melanoma cell migration.Macrophages in atherosclerosis: a dynamic balance.High density lipoprotein stimulated migration of macrophages depends on the scavenger receptor class B, type I, PDZK1 and Akt1 and is blocked by sphingosine 1 phosphate receptor antagonists.Atherosclerosis.A role for repressive complexes and H3K9 di-methylation in PRDM5-associated brittle cornea syndrome.Netrin-1 is a critical autocrine/paracrine factor for osteoclast differentiationDeep Transcriptomic Profiling of M1 Macrophages Lacking Trpc3.Macrophagic CD146 promotes foam cell formation and retention during atherosclerosisRhoB regulates the function of macrophages in the hypoxia-induced inflammatory response.The neuroimmune guidance cue netrin-1: a new therapeutic target in cardiovascular diseaseThe dynamic lives of macrophage and dendritic cell subsets in atherosclerosis.The regulation of pulmonary inflammation by the hypoxia-inducible factor-hydroxylase oxygen-sensing pathway.Potential contributions of intimal and plaque hypoxia to atherosclerosis.Hypoxia in Atherogenesis.Netrin-1 promoted pancreatic cancer cell proliferation by upregulation of Mdm2.HIF-mediated innate immune responses: cell signaling and therapeutic implications.Macrophages in vascular inflammation and atherosclerosis.Evaluation of Netrin-1 Levels and Albuminuria in Patients With Diabetes.HIF-1α and PFKFB3 Mediate a Tight Relationship Between Proinflammatory Activation and Anerobic Metabolism in Atherosclerotic Macrophages.Netrin-1 Regulates Fibrocyte Accumulation in the Decellularized Fibrotic Sclerodermatous Lung Microenvironment and in Bleomycin-Induced Pulmonary Fibrosis.Netrin-1 is highly expressed and required in inflammatory infiltrates in wear particle-induced osteolysis.Netrin-1 and its receptor Unc5b are novel targets for the treatment of inflammatory arthritis.The macrophage C-type lectin receptor CLEC5A (MDL-1) expression is associated with early plaque progression and promotes macrophage survival.Lentiviral-Mediated Netrin-1 Overexpression Improves Motor and Sensory Functions in SCT Rats Associated with SYP and GAP-43 Expressions.The role of monocytosis and neutrophilia in atherosclerosis.Guidance Molecules in Vascular Smooth Muscle
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P2860
Hypoxia induces netrin-1 and Unc5b in atherosclerotic plaques: mechanism for macrophage retention and survival
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 18 April 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Hypoxia induces netrin-1 and U ...... rophage retention and survival
@en
Hypoxia induces netrin-1 and U ...... ophage retention and survival.
@nl
type
label
Hypoxia induces netrin-1 and U ...... rophage retention and survival
@en
Hypoxia induces netrin-1 and U ...... ophage retention and survival.
@nl
prefLabel
Hypoxia induces netrin-1 and U ...... rophage retention and survival
@en
Hypoxia induces netrin-1 and U ...... ophage retention and survival.
@nl
P2093
P2860
P50
P921
P1476
Hypoxia induces netrin-1 and U ...... rophage retention and survival
@en
P2093
Bernd Hewing
Bhama Ramkhelawon
Jessica L Feig
Scott Oldebeken
P2860
P304
P356
10.1161/ATVBAHA.112.301008
P407
P577
2013-04-18T00:00:00Z