Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers - Wikidata - wikimedia - TriplyDB

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers

http://www.wikidata.org/entity/Q37225388

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Days to criterion as an indicator of toxicity associated with human Alzheimer amyloid-beta oligomers Molecular mechanisms of cognitive dysfunction following traumatic brain injury Comment on "ApoE-Directed Therapeutics Rapidly Clear -Amyloid and Reverse Deficits in AD Mouse Models"Dietary cholesterol degrades rabbit long term memory for discrimination learning but facilitates acquisition of discrimination reversal Opposing effects of Apoe/Apoa1 double deletion on amyloid-β pathology and cognitive performance in APP mice.Liver X receptor agonist treatment ameliorates amyloid pathology and memory deficits caused by high-fat diet in APP23 mice.The effects of cholesterol on learning and memory.Apolipoprotein A-I deficiency increases cerebral amyloid angiopathy and cognitive deficits in APP/PS1DeltaE9 mice Altered synaptic structure in the hippocampus in a mouse model of Alzheimer's disease with soluble amyloid-β oligomers and no plaque pathology Genome-wide alteration of histone H3K9 acetylation pattern in mouse offspring prenatally exposed to arsenic Early fear memory defects are associated with altered synaptic plasticity and molecular architecture in the TgCRND8 Alzheimer's disease mouse model.Improvement of memory deficits and amyloid-β clearance in aged APP23 mice treated with a combination of anti-amyloid-β antibody and LXR agonist Systematic review of the relationship between amyloid-β levels and measures of transgenic mouse cognitive deficit in Alzheimer's disease.Dietary Cholesterol Concentration and Duration Degrade Long-Term Memory of Classical Conditioning of the Rabbit's Nictitating Membrane Response.Liver X receptor agonist treatment significantly affects phenotype and transcriptome of APOE3 and APOE4 Abca1 haplo-deficient mice The isotropic fractionator provides evidence for differential loss of hippocampal neurons in two mouse models of Alzheimer's disease.Abca1 deficiency affects Alzheimer's disease-like phenotype in human ApoE4 but not in ApoE3-targeted replacement mice.The role of ATP-binding cassette transporter A1 in Alzheimer's disease and neurodegeneration Genome-wide approaches reveal EGR1-controlled regulatory networks associated with neurodegeneration.ABCA1 Deficiency Affects Basal Cognitive Deficits and Dendritic Density in Mice.ATP-binding cassette transporter A1: from metabolism to neurodegeneration.Proton pump inhibitor lansoprazole is a nuclear liver X receptor agonist.Differential regulation of ABCA1 and ABCG1 gene expressions in the remodeling mouse hippocampus after entorhinal cortex lesion and liver-X receptor agonist treatment Rhynchophylline Protects Against the Amyloid β-Induced Increase of Spontaneous Discharges in the Hippocampal CA1 Region of Rats.

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Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers

http://www.wikidata.org/entity/Q37225388

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 30 April 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers

@en

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers.

@nl

type

label

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers

@en

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers.

@nl

prefLabel

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers

@en

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers.

@nl

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P1476

Memory deficits in APP23/Abca1+/- mice correlate with the level of Aβ oligomers

@en

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Andrea Cronican

http://www.w3.org/2001/XMLSchema#string

Iliya Lefterov

http://www.w3.org/2001/XMLSchema#string

Nicholas F Fitz

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Preslav Lefterov

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Radosveta Koldamova

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P2860

Apolipoprotein A-I directly interacts with amyloid precursor protein and inhibits A beta aggregation and toxicity

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

ApoE promotes the proteolytic degradation of Abeta

The liver X receptor ligand T0901317 decreases amyloid beta production in vitro and in a mouse model of Alzheimer's disease

Apolipoprotein E isoforms and apolipoprotein AI protect from amyloid precursor protein carboxy terminal fragment-associated cytotoxicity

Deficiency of ABCA1 impairs apolipoprotein E metabolism in brain

Isoform-specific binding of apolipoprotein E to beta-amyloid

A specific amyloid-beta protein assembly in the brain impairs memory

Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis

Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

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scholarly article

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10.1042/AN20090015

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2

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1

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Matthias Staufenbiel

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2009-04-30T00:00:00Z

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19570031

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2695585

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