Implications for immunotherapy of tumor-mediated T-cell apoptosis associated with loss of the tumor suppressor PTEN in glioblastoma. - Wikidata - wikimedia - TriplyDB

Implications for immunotherapy of tumor-mediated T-cell apoptosis associated with loss of the tumor suppressor PTEN in glioblastoma.

Implications for immunotherapy of tumor-mediated T-cell apoptosis associated with loss of the tumor suppressor PTEN in glioblastoma.

http://www.wikidata.org/entity/Q37226831

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An integrative characterization of recurrent molecular aberrations in glioblastoma genomes.Immunocompetent murine models for the study of glioblastoma immunotherapy Human IP10-scFv and DC-induced CTL synergistically inhibit the growth of glioma in a xenograft model.Experimental research of host macrophage canceration induced by glioma stem progenitor cells Vaccine-based immunotherapy for glioblastoma.The future of glioblastoma therapy: synergism of standard of care and immunotherapy.Immune checkpoint inhibition and its relationship with hypermutation phenoytype as a potential treatment for Glioblastoma.Mechanisms of Intrinsic Tumor Resistance to Immunotherapy.

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Implications for immunotherapy of tumor-mediated T-cell apoptosis associated with loss of the tumor suppressor PTEN in glioblastoma.

http://www.wikidata.org/entity/Q37226831

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 September 2010

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Implications for immunotherapy ...... ppressor PTEN in glioblastoma.

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Implications for immunotherapy ...... ppressor PTEN in glioblastoma.

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Implications for immunotherapy ...... ppressor PTEN in glioblastoma.

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Implications for immunotherapy ...... ppressor PTEN in glioblastoma.

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Implications for immunotherapy ...... ppressor PTEN in glioblastoma.

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Implications for immunotherapy ...... ppressor PTEN in glioblastoma.

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J.JOCN.2010.04.021

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Journal of Clinical Neuroscience

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Implications for immunotherapy ...... uppressor PTEN in glioblastoma

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Andrew T Parsa

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James S Waldron

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Michael E Sughrue

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Russell O Pieper

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Seunggu Han

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Steven A Mills

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Tarik Tihan

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P2860

B7-H1, a third member of the B7 family, co-stimulates T-cell proliferation and interleukin-10 secretion

Tumor-associated B7-H1 promotes T-cell apoptosis: a potential mechanism of immune evasion

Cancer immunoediting: from immunosurveillance to tumor escape

Honokiol-mediated inhibition of PI3K/mTOR pathway: a potential strategy to overcome immunoresistance in glioma, breast, and prostate carcinoma without impacting T cell function

Loss of tumor suppressor PTEN function increases B7-H1 expression and immunoresistance in glioma

B7-H1 pathway and its role in the evasion of tumor immunity.

B7-homolog 1 expression by human glioma: a new mechanism of immune evasion.

Modulation of major histocompatibility complex Class I molecules and major histocompatibility complex-bound immunogenic peptides induced by interferon-alpha and interferon-gamma treatment of human glioblastoma multiforme.

In a model of tumor dormancy, long-term persistent leukemic cells have increased B7-H1 and B7.1 expression and resist CTL-mediated lysis.

Expression of the B7-related molecule B7-H1 by glioma cells: a potential mechanism of immune paralysis.

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apoptotic process

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