Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma. - Wikidata - wikimedia - TriplyDB

Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

http://www.wikidata.org/entity/Q37230411

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Nicotinamide metabolism regulates glioblastoma stem cell maintenance.Identification of a novel NAMPT inhibitor by CRISPR/Cas9 chemogenomic profiling in mammalian cells.An NAD+-dependent transcriptional program governs self-renewal and radiation resistance in glioblastoma MicroRNA Regulation of Glycolytic Metabolism in Glioblastoma.The Alkylating Chemotherapeutic Temozolomide Induces Metabolic Stress in IDH1-Mutant Cancers and Potentiates NAD+ Depletion-Mediated Cytotoxicity.The Glycolytic Switch in Tumors: How Many Players Are Involved?Chromosomal instability induced by increased BIRC5/Survivin levels affects tumorigenicity of glioma cells.Targeting Metabolism for Cancer Therapy.Rapid progression to glioblastoma in a subset of IDH-mutated astrocytomas: a genome-wide analysis.Failure of the PTEN/aPKC/Lgl Axis Primes Formation of Adult Brain Tumours in Drosophila.

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Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

http://www.wikidata.org/entity/Q37230411

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 13 April 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

@da

vědecký článek

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name

Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

@en

Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

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type

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Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

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Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

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prefLabel

Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

@en

Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

@nl

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1078-0432.CCR-15-2274

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Clinical Cancer Research

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Myc-Driven Glycolysis Is a Therapeutic Target in Glioblastoma.

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A John Iafrate

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Andrew S Chi

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Daniel P Cahill

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Hiroaki Wakimoto

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Kensuke Tateishi

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Maristela L Onozato

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Nina Lelic

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Quan Ho

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Sudhandra Sundaram

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Tracy T Batchelor

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P2860

An integrated genomic analysis of human glioblastoma multiforme

Glucose deprivation contributes to the development of KRAS pathway mutations in tumor cells

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

Loss of NAPRT1 expression by tumor-specific promoter methylation provides a novel predictive biomarker for NAMPT inhibitors.

Comprehensive, Integrative Genomic Analysis of Diffuse Lower-Grade Gliomas.

BET bromodomain inhibition as a therapeutic strategy to target c-Myc

The somatic genomic landscape of glioblastoma

Metabolomics analysis of metabolic effects of nicotinamide phosphoribosyltransferase (NAMPT) inhibition on human cancer cells

A unique glucose-dependent apoptotic pathway induced by c-Myc

The landscape of somatic copy-number alteration across human cancers.

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4452-4465

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scholarly article

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10.1158/1078-0432.CCR-15-2274

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2016-04-13T00:00:00Z

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27076630

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5010492

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