Tie2cre-induced inactivation of the miRNA-processing enzyme Dicer disrupts invariant NKT cell development. - Wikidata - wikimedia - TriplyDB

Tie2cre-induced inactivation of the miRNA-processing enzyme Dicer disrupts invariant NKT cell development.

Tie2cre-induced inactivation of the miRNA-processing enzyme Dicer disrupts invariant NKT cell development.

http://www.wikidata.org/entity/Q37238805

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Lack of an association of miR-938 SNP in IDDM10 with human type 1 diabetes The adaptor protein Shc integrates growth factor and ECM signaling during postnatal angiogenesis.miR-150 regulates the development of NK and iNKT cells.JAK2V617F-positive endothelial cells contribute to clotting abnormalities in myeloproliferative neoplasms.Niacin ameliorates ulcerative colitis via prostaglandin D2-mediated D prostanoid receptor 1 activation.Next-generation sequencing identifies the natural killer cell microRNA transcriptome.Targeted deletion of Dicer from proximal tubules protects against renal ischemia-reperfusion injury.MicroRNA miR-125a controls hematopoietic stem cell number Distinct requirements of microRNAs in NK cell activation, survival, and function Identification of mouse serum miRNA endogenous references by global gene expression profiles.Dicer in immune cell development and function.miR-22 controls Irf8 mRNA abundance and murine dendritic cell development.MicroRNA control of lymphocyte differentiation and function Let-7 microRNAs target the lineage-specific transcription factor PLZF to regulate terminal NKT cell differentiation and effector function.MicroRNA-processing enzyme Dicer is required in epicardium for coronary vasculature development.Genetic control of murine invariant natural killer T-cell development dynamically differs dependent on the examined tissue type.MicroRNA-301a regulation of a T-helper 17 immune response controls autoimmune demyelination Stop the dicing in hematopoiesis: what have we learned?Histone deacetylases inhibitor Trichostatin A ameliorates DNFB-induced allergic contact dermatitis and reduces epidermal Langerhans cells in mice Lack of microRNA miR-150 reduces the capacity of epidermal Langerhans cell cross-presentation Aging affects epidermal Langerhans cell development and function and alters their miRNA gene expression profile.Regulation of lipid metabolism by Dicer revealed through SILAC mice Critical role for miR-181a/b-1 in agonist selection of invariant natural killer T cells Aging-induced dysregulation of dicer1-dependent microRNA expression impairs angiogenic capacity of rat cerebromicrovascular endothelial cells.Deletion of microRNA miR-223 increases Langerhans cell cross-presentation.TGF-β1-induced transcription factor networks in Langerhans cell development and maintenance.miR-17∼92 family clusters control iNKT cell ontogenesis via modulation of TGF-β signaling.Mechanistic target of rapamycin complex 1 is critical for invariant natural killer T-cell development and effector function.Raising the NKT cell family.MiR-34a promotes DCs development and inhibits their function on T cell activation by targeting WNT1.Loss of microRNAs in thymus perturbs invariant NKT cell development and function MicroRNAs and Multiple Sclerosis.MicroRNAs are key regulators controlling iNKT and regulatory T-cell development and function.MicroRNA in immunity and autoimmunity.Immuno-miRs: critical regulators of T-cell development, function and ageing.Roles of microRNAs in psoriasis: Immunological functions and potential biomarkers.Overexpression of Vα14Jα18 TCR promotes development of iNKT cells in the absence of miR-181a/b-1.Deletion of miRNAs in bone marrow prevents streptozotocin-induced murine autoimmune diabetes but deletion of miR-155 does not.MicroRNA expression profiling identifies potential serum biomarkers for non-segmental vitiligo.Fabp4-Cre-mediated deletion of the miRNA-processing enzyme Dicer causes mouse embryonic lethality.

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Tie2cre-induced inactivation of the miRNA-processing enzyme Dicer disrupts invariant NKT cell development.

http://www.wikidata.org/entity/Q37238805

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 June 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Tie2cre-induced inactivation o ...... nvariant NKT cell development.

@en

Tie2cre-induced inactivation o ...... nvariant NKT cell development.

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type

label

Tie2cre-induced inactivation o ...... nvariant NKT cell development.

@en

Tie2cre-induced inactivation o ...... nvariant NKT cell development.

@nl

prefLabel

Tie2cre-induced inactivation o ...... nvariant NKT cell development.

@en

Tie2cre-induced inactivation o ...... nvariant NKT cell development.

@nl

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PNAS.0811119106

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Tie2cre-induced inactivation o ...... nvariant NKT cell development.

@en

P2093

Chang-Gui Li

http://www.w3.org/2001/XMLSchema#string

Dong-Mei Meng

http://www.w3.org/2001/XMLSchema#string

Hong-Zhi He

http://www.w3.org/2001/XMLSchema#string

Jianrui Xu

http://www.w3.org/2001/XMLSchema#string

Jin-Xiong She

http://www.w3.org/2001/XMLSchema#string

Kook-Heon Seo

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Li Zhou

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Qing-Sheng Mi

http://www.w3.org/2001/XMLSchema#string

Rafal Pacholczyk

http://www.w3.org/2001/XMLSchema#string

Zheng Dong

http://www.w3.org/2001/XMLSchema#string

P2860

The RNaseIII enzyme Dicer is required for morphogenesis but not patterning of the vertebrate limb.

MicroRNA targets in immune genes and the Dicer/Argonaute and ARE machinery components

Intravascular immune surveillance by CXCR6+ NKT cells patrolling liver sinusoids

MicroRNAs: genomics, biogenesis, mechanism, and function

The biology of NKT cells

NKT cells derive from double-positive thymocytes that are positively selected by CD1d

Dicer is essential for mouse development

Hypersusceptibility to vesicular stomatitis virus infection in Dicer1-deficient mice is due to impaired miR24 and miR93 expression

GATA-3 regulates the development and function of invariant NKT cells

A role for Dicer in immune regulation

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10266-10271

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scholarly article

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10.1073/PNAS.0811119106

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25

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106

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2009-06-09T00:00:00Z

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26278906

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19509335

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2700920

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