Modulation of Helicobacter pylori induced interleukin-8 synthesis in gastric epithelial cells mediated by cag PAI encoded VirD4 homologue
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Type IV secretion: intercellular transfer of macromolecules by systems ancestrally related to conjugation machinesHost cell responses to genotypically similar Helicobacter pylori isolates from United States and Japan.Role of Helicobacter pylori cag region genes in colonization and gastritis in two animal modelsComparison of genetic divergence and fitness between two subclones of Helicobacter pyloriIV. Helicobacter pylori strain-specific activation of signal transduction cascades related to gastric inflammation.Helicobacter pylori genetic diversity and risk of human disease.TraG-like proteins of DNA transfer systems and of the Helicobacter pylori type IV secretion system: inner membrane gate for exported substrates?Insights into the Role of Helicobacter pylori Infection in Preeclampsia: From the Bench to the Bedsidepathogenesis of Helicobacter pylori-induced gastric inflammation.DNA-level characterization of Helicobacter pylori strains from patients with overt disease and with benign infections in BangladeshRelation of CagA seropositivity to cagPAI phenotype and histological grade of gastritis in patients with Helicobacter pylori infection.Functional analysis of the cag pathogenicity island in Helicobacter pylori isolates from patients with gastritis, peptic ulcer, and gastric cancer.Roles of virD4 and cagG genes in the cag pathogenicity island of Helicobacter pylori using a Mongolian gerbil modelAssociation between cag-pathogenicity island in Helicobacter pylori isolates from peptic ulcer, gastric carcinoma, and non-ulcer dyspepsia subjects with histological changesAntibodies anti-CagA cross-react with trophoblast cells: a risk factor for pre-eclampsia?Helicobacter pylori, Chlamydiae pneumoniae and trachomatis as probable etiological agents of preeclampsia.Helicobacter pylori infection contributes to placental impairment in preeclampsia: basic and clinical evidences.Functional analysis of the Helicobacter pylori cag pathogenicity island reveals both VirD4-CagA-dependent and VirD4-CagA-independent mechanisms.cag+ Helicobacter pylori induces homotypic aggregation of macrophage-like cells by up-regulation and recruitment of intracellular adhesion molecule 1 to the cell surface.Src is the kinase of the Helicobacter pylori CagA protein in vitro and in vivo.Current Helicobacter pylori infection is significantly associated with subclinical coronary atherosclerosis in healthy subjects: A cross-sectional study.
P2860
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P2860
Modulation of Helicobacter pylori induced interleukin-8 synthesis in gastric epithelial cells mediated by cag PAI encoded VirD4 homologue
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1999
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Modulation of Helicobacter pyl ...... ag PAI encoded VirD4 homologue
@en
Modulation of Helicobacter pyl ...... g PAI encoded VirD4 homologue.
@nl
type
label
Modulation of Helicobacter pyl ...... ag PAI encoded VirD4 homologue
@en
Modulation of Helicobacter pyl ...... g PAI encoded VirD4 homologue.
@nl
prefLabel
Modulation of Helicobacter pyl ...... ag PAI encoded VirD4 homologue
@en
Modulation of Helicobacter pyl ...... g PAI encoded VirD4 homologue.
@nl
P2093
P2860
P356
P1476
Modulation of Helicobacter pyl ...... ag PAI encoded VirD4 homologue
@en
P2093
P2860
P304
P356
10.1136/JCP.52.9.653
P407
P577
1999-09-01T00:00:00Z