Pathogenic CD4⁺ T cells recognizing an unstable peptide of insulin are directly recruited into islets bypassing local lymph nodes.
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Beta cell antigens in type 1 diabetes: triggers in pathogenesis and therapeutic targetsMHC Class II Auto-Antigen Presentation is UnconventionalThe central role of antigen presentation in islets of Langerhans in autoimmune diabetesZnT8-reactive T cells are weakly pathogenic in NOD mice but can participate in diabetes under inflammatory conditionsA minor subset of Batf3-dependent antigen-presenting cells in islets of Langerhans is essential for the development of autoimmune diabetes.Beta cells transfer vesicles containing insulin to phagocytes for presentation to T cells.Class-switched anti-insulin antibodies originate from unconventional antigen presentation in multiple lymphoid sites.Antigen presentation events during the initiation of autoimmune diabetes in the NOD mouseThe role of islet antigen presenting cells and the presentation of insulin in the initiation of autoimmune diabetes in the NOD mouse.Lymphocyte repertoire selection and intracellular self/non-self-discrimination: historical overview.The ins and outs of MHC class II-mediated antigen processing and presentation.Proteolysis by Granzyme B Enhances Presentation of Autoantigenic Peptidylarginine Deiminase 4 Epitopes in Rheumatoid Arthritis.Macrophages and dendritic cells in islets of Langerhans in diabetic autoimmunity: a lesson on cell interactions in a mini-organ.Autoreactive helper T cells alleviate the need for intrinsic TLR signaling in autoreactive B cell activation.Bridge between type 1 diabetes in mouse and man.Type 1 diabetes induction in humanized mice.Autoreactive T cells in type 1 diabetes.Understanding Autoimmune Diabetes through the Prism of the Tri-Molecular Complex.MHC-mismatched mixed chimerism restores peripheral tolerance of noncross-reactive autoreactive T cells in NOD mice.Revealing the specificity of regulatory T cells in murine autoimmune diabetes.Pancreatic islets communicate with lymphoid tissues via exocytosis of insulin peptides
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Pathogenic CD4⁺ T cells recognizing an unstable peptide of insulin are directly recruited into islets bypassing local lymph nodes.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 14 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Pathogenic CD4⁺ T cells recogn ...... s bypassing local lymph nodes.
@en
Pathogenic CD4⁺ T cells recogn ...... s bypassing local lymph nodes.
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type
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Pathogenic CD4⁺ T cells recogn ...... s bypassing local lymph nodes.
@en
Pathogenic CD4⁺ T cells recogn ...... s bypassing local lymph nodes.
@nl
prefLabel
Pathogenic CD4⁺ T cells recogn ...... s bypassing local lymph nodes.
@en
Pathogenic CD4⁺ T cells recogn ...... s bypassing local lymph nodes.
@nl
P2860
P356
P1476
Pathogenic CD4⁺ T cells recogn ...... ts bypassing local lymph nodes
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P2093
James F Mohan
Mark S Anderson
P2860
P304
P356
10.1084/JEM.20130582
P407
P577
2013-10-14T00:00:00Z