Variation in antagonism of the interferon response to rotavirus NSP1 results in differential infectivity in mouse embryonic fibroblasts
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MAVS protein is attenuated by rotavirus nonstructural protein 1Rotavirus nonstructural protein 1 suppresses virus-induced cellular apoptosis to facilitate viral growth by activating the cell survival pathways during early stages of infectionInflammatory and oxidative stress in rotavirus infectionThe battle between rotavirus and its host for control of the interferon signaling pathwayThe Rotavirus Interferon Antagonist NSP1: Many Targets, Many QuestionsRotavirus structural proteins and dsRNA are required for the human primary plasmacytoid dendritic cell IFNalpha responseDiversity of interferon antagonist activities mediated by NSP1 proteins of different rotavirus strains.Characterization of rotavirus RNAs that activate innate immune signaling through the RIG-I-like receptors.The early interferon response to rotavirus is regulated by PKR and depends on MAVS/IPS-1, RIG-I, MDA-5, and IRF3.Molecular epidemiology and genetic evolution of the whole genome of G3P[8] human rotavirus in Wuhan, China, from 2000 through 2013Rotavirus antagonism of the innate immune responseExperimental Adaptation of Rotaviruses to Tumor Cell Lines.Rhesus rotavirus VP4 sequence-specific activation of mononuclear cells is associated with cholangiopathy in murine biliary atresia.Uniformity of rotavirus strain nomenclature proposed by the Rotavirus Classification Working Group (RCWG)Rotavirus immune responses and correlates of protection.The spike protein VP4 defines the endocytic pathway used by rotavirus to enter MA104 cells.Rotavirus NSP1 mediates degradation of interferon regulatory factors through targeting of the dimerization domainRotavirus NSP1 protein inhibits interferon-mediated STAT1 activation.Rotaviruses reach late endosomes and require the cation-dependent mannose-6-phosphate receptor and the activity of cathepsin proteases to enter the cell.Antigenemia, RNAemia, and innate immunity in children with acute rotavirus diarrhea.IRF3 inhibition by rotavirus NSP1 is host cell and virus strain dependent but independent of NSP1 proteasomal degradation.Selective inhibition of host cell signaling for rotavirus antivirals: PI3K/Akt/mTOR-mediated rotavirus pathogenesis.Roles of VP4 and NSP1 in determining the distinctive replication capacities of simian rotavirus RRV and bovine rotavirus UK in the mouse biliary tract.Permissive replication of homologous murine rotavirus in the mouse intestine is primarily regulated by VP4 and NSP1.Basal interferon signaling and therapeutic use of interferons in controlling rotavirus infection in human intestinal cells and organoids.
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Variation in antagonism of the interferon response to rotavirus NSP1 results in differential infectivity in mouse embryonic fibroblasts
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 06 May 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Variation in antagonism of the ...... in mouse embryonic fibroblasts
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Variation in antagonism of the ...... n mouse embryonic fibroblasts.
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type
label
Variation in antagonism of the ...... in mouse embryonic fibroblasts
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Variation in antagonism of the ...... n mouse embryonic fibroblasts.
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prefLabel
Variation in antagonism of the ...... in mouse embryonic fibroblasts
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Variation in antagonism of the ...... n mouse embryonic fibroblasts.
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P2093
P2860
P356
P1433
P1476
Variation in antagonism of the ...... in mouse embryonic fibroblasts
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P2093
P2860
P304
P356
10.1128/JVI.00585-09
P407
P577
2009-05-06T00:00:00Z