Chromatin modifications associated with DNA double-strand breaks repair as potential targets for neurological diseases.
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Prion-induced neurotoxicity: Possible role for cell cycle activity and DNA damage responseExtracellular vesicles as trans-genomic agents: Emerging roles in disease and evolution.Oxidative stress and its significant roles in neurodegenerative diseases and cancer.Poly(ADP-ribose) polymerase 1 is a novel target to promote axonal regeneration.Epigenetic drug discovery for Alzheimer's disease.NRAGE is involved in homologous recombination repair to resist the DNA-damaging chemotherapy and composes a ternary complex with RNF8-BARD1 to promote cell survival in squamous esophageal tumorigenesis.Looking above but not beyond the genome for therapeutics in neurology and psychiatry: epigenetic proteins and RNAs find a new focus.
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Chromatin modifications associated with DNA double-strand breaks repair as potential targets for neurological diseases.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Chromatin modifications associ ...... ets for neurological diseases.
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Chromatin modifications associ ...... ets for neurological diseases.
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type
label
Chromatin modifications associ ...... ets for neurological diseases.
@en
Chromatin modifications associ ...... ets for neurological diseases.
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prefLabel
Chromatin modifications associ ...... ets for neurological diseases.
@en
Chromatin modifications associ ...... ets for neurological diseases.
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P2860
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Chromatin modifications associ ...... ets for neurological diseases.
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Camille Brochier
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10.1007/S13311-013-0210-9
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2013-10-01T00:00:00Z