Injury, inflammation and the emergence of human-specific genes.
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Biochemical, Cellular, Physiological, and Pathological Consequences of Human Loss of N-Glycolylneuraminic Acid.Loss of CMAH during Human Evolution Primed the Monocyte-Macrophage Lineage toward a More Inflammatory and Phagocytic State.Mice engrafted with human hematopoietic stem cells support a human myeloid cell inflammatory response in vivo.Imaging mass spectrometry for accessing molecular changes during burn wound healing.
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Injury, inflammation and the emergence of human-specific genes.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 13 February 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Injury, inflammation and the emergence of human-specific genes.
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Injury, inflammation and the emergence of human-specific genes.
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Injury, inflammation and the emergence of human-specific genes.
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Injury, inflammation and the emergence of human-specific genes.
@en
Injury, inflammation and the emergence of human-specific genes.
@en-gb
Injury, inflammation and the emergence of human-specific genes.
@nl
prefLabel
Injury, inflammation and the emergence of human-specific genes.
@en
Injury, inflammation and the emergence of human-specific genes.
@en-gb
Injury, inflammation and the emergence of human-specific genes.
@nl
P2860
P356
P1476
Injury, inflammation and the emergence of human-specific genes.
@en
P2093
Raul Coimbra
Todd Costantini
P2860
P304
P356
10.1111/WRR.12422
P577
2016-02-13T00:00:00Z