Preferential HIV infection of CCR6+ Th17 cells is associated with higher levels of virus receptor expression and lack of CCR5 ligands.
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Sex hormones selectively impact the endocervical mucosal microenvironment: implications for HIV transmissionGlucose Metabolism in T Cells and Monocytes: New Perspectives in HIV PathogenesisNew Insights about Treg and Th17 Cells in HIV Infection and Disease ProgressionInduction of intestinal immunity by mucosal vaccines as a means of controlling HIV infectionRectal HSV-2 Infection May Increase Rectal SIV Acquisition Even in the Context of SIVΔnef VaccinationCellular superspreaders: an epidemiological perspective on HIV infection inside the bodyHuman Beta Defensin 2 Selectively Inhibits HIV-1 in Highly Permissive CCR6⁺CD4⁺ T Cells.The loss of CCR6+ and CD161+ CD4+ T-cell homeostasis contributes to disease progression in SIV-infected rhesus macaques.Defective IL-17- and IL-22-dependent mucosal host response to Candida albicans determines susceptibility to oral candidiasis in mice expressing the HIV-1 transgene.Low SAMHD1 expression following T-cell activation and proliferation renders CD4+ T cells susceptible to HIV-1.Impaired Th17 polarization of phenotypically naive CD4(+) T-cells during chronic HIV-1 infection and potential restoration with early ARTSchistosoma mansoni Infection in Ugandan Men Is Associated with Increased Abundance and Function of HIV Target Cells in Blood, but Not the Foreskin: A Cross-sectional Study.Integrin α4β7 Expression Increases HIV Susceptibility in Activated Cervical CD4+ T Cells by an HIV Attachment-Independent Mechanism.HCV Specific IL-21 Producing T Cells but Not IL-17A Producing T Cells Are Associated with HCV Viral Control in HIV/HCV CoinfectionSequential Dysfunction and Progressive Depletion of Candida albicans-Specific CD4 T Cell Response in HIV-1 Infection.HIV persistence in the setting of antiretroviral therapy: when, where and how does HIV hide?Gut Mucosal Barrier Dysfunction, Microbial Dysbiosis, and Their Role in HIV-1 Disease Progression.Chemokine Levels in the Penile Coronal Sulcus Correlate with HIV-1 Acquisition and Are Reduced by Male Circumcision in Rakai, Uganda.Th1/17 Polarization of CD4 T Cells Supports HIV-1 Persistence during Antiretroviral Therapy.The transcriptome of HIV-1 infected intestinal CD4+ T cells exposed to enteric bacteria.Identification of novel HIV-1 dependency factors in primary CCR4(+)CCR6(+)Th17 cells via a genome-wide transcriptional approach.Th17 Cells Are Preferentially Infected Very Early after Vaginal Transmission of SIV in Macaques.Persistence of integrated HIV DNA in CXCR3 + CCR6 + memory CD4+ T cells in HIV-infected individuals on antiretroviral therapy.Human Immunodeficiency Virus Immune Cell Receptors, Coreceptors, and Cofactors: Implications for Prevention and Treatment.Human Th17 Cells Lack HIV-Inhibitory RNases and Are Highly Permissive to Productive HIV Infection.HIV persists in CCR6+CD4+ T cells from colon and blood during antiretroviral therapy.Impairment of CCR6+ and CXCR3+ Th Cell Migration in HIV-1 Infection Is Rescued by Modulating Actin Polymerization.Inflammation and HIV Transmission in Sub-Saharan Africa.Mass Cytometric Analysis of HIV Entry, Replication, and Remodeling in Tissue CD4+ T CellsToll-Like Receptor 2 Ligation Enhances HIV-1 Replication in Activated CCR6+ CD4+ T Cells by Increasing Virus Entry and Establishing a More Permissive Environment to Infection.Paediatric HIV infection: the potential for cure.HIV-TB co-infection: mechanisms that drive reactivation of Mycobacterium tuberculosis in HIV infection.Penile Anaerobic Dysbiosis as a Risk Factor for HIV Infection.No Evidence for Association of β-Defensin Genomic Copy Number with HIV Susceptibility, HIV Load during Clinical Latency, or Progression to AIDS.Vector order determines protection against pathogenic simian immunodeficiency virus infection in a triple component vaccine by balancing CD4+ and CD8+ T-cell responses.HIV-1 selectively targets gut-homing CCR6+CD4+ T cells via mTOR-dependent mechanisms.Unique microRNA expression in the colonic mucosa during chronic HIV-1 infection.New insights into the heterogeneity of Th17 subsets contributing to HIV-1 persistence during antiretroviral therapyDifferential effects of HIV transmission from monocyte-derived dendritic cells vs. monocytes to IL-17+CD4+ T cells.Parallel assessment of Th17 cell frequencies by surface marker co-expression versus ex vivo IL-17 production in HIV-1 infection.
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Preferential HIV infection of CCR6+ Th17 cells is associated with higher levels of virus receptor expression and lack of CCR5 ligands.
description
article científic
@ca
article scientifique
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articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 31 July 2013
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Preferential HIV infection of ...... sion and lack of CCR5 ligands.
@en
Preferential HIV infection of ...... sion and lack of CCR5 ligands.
@nl
type
label
Preferential HIV infection of ...... sion and lack of CCR5 ligands.
@en
Preferential HIV infection of ...... sion and lack of CCR5 ligands.
@nl
prefLabel
Preferential HIV infection of ...... sion and lack of CCR5 ligands.
@en
Preferential HIV infection of ...... sion and lack of CCR5 ligands.
@nl
P2093
P2860
P356
P1433
P1476
Preferential HIV infection of ...... sion and lack of CCR5 ligands.
@en
P2093
Catarina E Hioe
Fatima Nawaz
Guomiao Shen
James Arthos
Martin J Wolff
Michael A Poles
Michael Tuen
Yelina Alvarez
P2860
P304
10843-10854
P356
10.1128/JVI.01838-13
P407
P577
2013-07-31T00:00:00Z