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Reactive oxygen species controls endometriosis progression.

Reactive oxygen species controls endometriosis progression.

http://www.wikidata.org/entity/Q37257155

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Nutritional aspects related to endometriosis Defining future directions for endometriosis research: workshop report from the 2011 World Congress of Endometriosis In Montpellier, France The effects of oxidative stress on female reproduction: a review Mitochondrial genome variations in advanced stage endometriosis: a study in South Indian population ARID1A Expression is Down-Regulated by Oxidative Stress in Endometriosis and Endometriosis-Associated Ovarian Cancer Is the oocyte quality affected by endometriosis? A review of the literature Simvastatin induces apoptosis and alters cytoskeleton in endometrial stromal cells ABO and Rhesus blood groups and risk of endometriosis in a French Caucasian population of 633 patients living in the same geographic area.An update on the pharmacological management of endometriosis.Antiproliferative effects of cannabinoid agonists on deep infiltrating endometriosis.The distinguishing cellular and molecular features of the endometriotic ovarian cyst: from pathophysiology to the potential endometrioma-mediated damage to the ovary.A pilot study to search possible mechanisms of ultralong gonadotropin-releasing hormone agonist therapy in IVF-ET patients with endometriosis Differential immune and genetic responses in rat models of Crohn's colitis and ulcerative colitis.Increased serum oxidative stress markers in women with uterine leiomyoma Low birth weight is strongly associated with the risk of deep infiltrating endometriosis: results of a 743 case-control study.The mTOR/AKT inhibitor temsirolimus prevents deep infiltrating endometriosis in mice.Soluble ligands for the NKG2D receptor are released during endometriosis and correlate with disease severity.Infertility and chylous ascites? A case report.Urine, peritoneal fluid and omental fat proteomes of reproductive age women: Endometriosis-related changes and associations with endocrine disrupting chemicals.Pathophysiology of Endometriosis: Role of High Mobility Group Box-1 and Toll-Like Receptor 4 Developing Inflammation in Endometrium.A promise in the treatment of endometriosis: an observational cohort study on ovarian endometrioma reduction by N-acetylcysteine Functional expression of aryl hydrocarbon receptor on mast cells populating human endometriotic tissues.Oxidative Stress Statues in Serum and Follicular Fluid of Women with Endometriosis.Ovarian-like differentiation in eutopic and ectopic endometrioses with aberrant FSH receptor, INSL3 and GATA4/6 expression PI3K-Akt-mTOR and MAPK signaling pathways in polycystic ovarian syndrome, uterine leiomyomas and endometriosis: an update.New treatment strategies and emerging drugs in endometriosis.Oxidative stress biomarkers in patients with endometriosis: systematic review.Novel insights on the malignant transformation of endometriosis into ovarian carcinoma.Pathogenetic Mechanisms of Deep Infiltrating Endometriosis.Targeting oxidative stress to treat endometriosis.MAP kinases and the inflammatory signaling cascade as targets for the treatment of endometriosis?Oxidative stress and oocyte quality: ethiopathogenic mechanisms of minimal/mild endometriosis-related infertility.Kinase signalling pathways in endometriosis: potential targets for non-hormonal therapeutics.Dysregulation of the ADAM17/Notch signalling pathways in endometriosis: from oxidative stress to fibrosis.Arterial stiffness is increased in young women with endometriosis.Protein oxidative stress markers in peritoneal fluids of women with deep infiltrating endometriosis are increased.Antifibrotic properties of epigallocatechin-3-gallate in endometriosis.Surgery versus pharmacological treatment for endometriosis.Epithelial-to-mesenchymal transition in the development of endometriosis.Caveolin-1 sensitizes cisplatin-induced lung cancer cell apoptosis via superoxide anion-dependent mechanism.

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Reactive oxygen species controls endometriosis progression.

http://www.wikidata.org/entity/Q37257155

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 04 June 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Reactive oxygen species controls endometriosis progression.

@en

Reactive oxygen species controls endometriosis progression.

@nl

type

label

Reactive oxygen species controls endometriosis progression.

@en

Reactive oxygen species controls endometriosis progression.

@nl

prefLabel

Reactive oxygen species controls endometriosis progression.

@en

Reactive oxygen species controls endometriosis progression.

@nl

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AJPATH.2009.080804

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The American Journal of Pathology

P1476

Reactive oxygen species controls endometriosis progression.

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P2093

Bernard Weill

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Carole Nicco

http://www.w3.org/2001/XMLSchema#string

Charlotte Ngô

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Christiane Chéreau

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Frédéric Batteux

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P2860

Cell transformation by the superoxide-generating oxidase Mox1

Hydrogen peroxide mediates the cell growth and transformation caused by the mitogenic oxidase Nox1

Superoxide dismutase: improved assays and an assay applicable to acrylamide gels

Catalase in vitro

Production of large amounts of hydrogen peroxide by human tumor cells

Oxidative stress may be a piece in the endometriosis puzzle.

Angiogenesis: a new theory for endometriosis.

ROS-generating mitochondrial DNA mutations can regulate tumor cell metastasis.

Role of endometriosis in cancer and tumor development.

Experimental endometriosis: the nude mouse as a xenographic host.

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225-234

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scholarly article

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10.2353/AJPATH.2009.080804

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1

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175

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Charles Chapron

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2009-06-04T00:00:00Z

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26268227

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19498006

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