Up-regulation of soluble Axl and Mer receptor tyrosine kinases negatively correlates with Gas6 in established multiple sclerosis lesions - Wikidata - wikimedia - TriplyDB

Up-regulation of soluble Axl and Mer receptor tyrosine kinases negatively correlates with Gas6 in established multiple sclerosis lesions

Up-regulation of soluble Axl and Mer receptor tyrosine kinases negatively correlates with Gas6 in established multiple sclerosis lesions

http://www.wikidata.org/entity/Q37257176

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The Gas6/TAM System and Multiple Sclerosis Ligand Activation of TAM Family Receptors-Implications for Tumor Biology and Therapeutic Response Polymorphisms in the receptor tyrosine kinase MERTK gene are associated with multiple sclerosis susceptibility Association of plasma levels of Protein S with disease severity in multiple sclerosis.ADAM-17: the enzyme that does it all.GAS6 enhances repair following cuprizone-induced demyelination.Gas6 increases myelination by oligodendrocytes and its deficiency delays recovery following cuprizone-induced demyelination TAM receptor tyrosine kinases: expression, disease and oncogenesis in the central nervous system.Targeted GAS6 delivery to the CNS protects axons from damage during experimental autoimmune encephalomyelitis Axl as a mediator of cellular growth and survival GAS6 receptor status is associated with dormancy and bone metastatic tumor formation Loss of the receptor tyrosine kinase Axl leads to enhanced inflammation in the CNS and delayed removal of myelin debris during experimental autoimmune encephalomyelitis Induction of a common microglia gene expression signature by aging and neurodegenerative conditions: a co-expression meta-analysis.Primary myeloma interaction and growth in coculture with healthy donor hematopoietic bone marrow.Common and Low Frequency Variants in MERTK Are Independently Associated with Multiple Sclerosis Susceptibility with Discordant Association Dependent upon HLA-DRB1*15:01 Status.Oligodendrocyte Regeneration and CNS Remyelination Require TACE/ADAM17 Growth arrest specific gene 6 protein concentration in cerebrospinal fluid correlates with relapse severity in multiple sclerosis.Growth arrest-specific protein 6 attenuates neutrophil migration and acute lung injury in sepsis Phagocyte dysfunction, tissue aging and degeneration.Receptor Tyrosine Kinase and Tyrosine Kinase Inhibitors: New Hope for Success in Multiple Sclerosis Therapy Axl and Mer Receptor Tyrosine Kinases: Distinct and Nonoverlapping Roles in Inflammation and Cancer?Key Roles of AXL and MER Receptor Tyrosine Kinases in Resistance to Multiple Anticancer Therapies.Diminished soluble levels of growth arrest specific protein 6 and tyrosine kinase receptor Axl in patients with rheumatoid arthritis.New candidates for CD4 T cell pathogenicity in experimental neuroinflammation and multiple sclerosis.A Janus role for MerTK in the outcome of septic shock.Erythrocyte efferocytosis modulates macrophages towards recovery after intracerebral hemorrhage.Loss of Gas6 and Axl signaling results in extensive axonal damage, motor deficits, prolonged neuroinflammation, and less remyelination following cuprizone exposure.Soluble TAM receptor tyrosine kinases in rheumatoid arthritis: correlation with disease activity and bone destruction.Antiphospholipid Antibodies Inhibit Trophoblast Toll-like Receptor and Inflammasome Negative Regulators.Gas6 as a putative noninvasive biomarker of hepatic fibrosis.The role of TAM family receptors and ligands in the nervous system: From development to pathobiology.Emerging PET Radiotracers and Targets for Imaging of Neuroinflammation in Neurodegenerative Diseases: Outlook Beyond TSPO Orthologous proteins of experimental de- and remyelination are differentially regulated in the CSF proteome of multiple sclerosis subtypes

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Up-regulation of soluble Axl and Mer receptor tyrosine kinases negatively correlates with Gas6 in established multiple sclerosis lesions

http://www.wikidata.org/entity/Q37257176

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 18 June 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Up-regulation of soluble Axl a ...... hed multiple sclerosis lesions

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Up-regulation of soluble Axl a ...... ed multiple sclerosis lesions.

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type

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Up-regulation of soluble Axl a ...... hed multiple sclerosis lesions

@en

Up-regulation of soluble Axl a ...... ed multiple sclerosis lesions.

@nl

prefLabel

Up-regulation of soluble Axl a ...... hed multiple sclerosis lesions

@en

Up-regulation of soluble Axl a ...... ed multiple sclerosis lesions.

@nl

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AJPATH.2009.080807

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The American Journal of Pathology

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Up-regulation of soluble Axl a ...... hed multiple sclerosis lesions

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Bridget Shafit-Zagardo

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Cedric S Raine

http://www.w3.org/2001/XMLSchema#string

Jason G Weinger

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Kakuri M Omari

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Kurt Marsden

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P2860

The protein encoded by a growth arrest-specific gene (gas6) is a new member of the vitamin K-dependent proteins related to protein S, a negative coregulator in the blood coagulation cascade

Axl receptor tyrosine kinase stimulated by the vitamin K-dependent protein encoded by growth-arrest-specific gene 6

Reevaluation of the roles of protein S and Gas6 as ligands for the receptor tyrosine kinase Rse/Tyro 3

Gas6/Axl signaling activates the phosphatidylinositol 3-kinase/Akt1 survival pathway to protect oligodendrocytes from tumor necrosis factor alpha-induced apoptosis

RETRACTED: A promiscuous liaison between IL-15 receptor and Axl receptor tyrosine kinase in cell death control.

Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Ligand recognition and homophilic interactions in Tyro3: structural insights into the Axl/Tyro3 receptor tyrosine kinase family

Tumor cell interactions with the extracellular matrix during invasion and metastasis

The transmembrane CXC-chemokine ligand 16 is induced by IFN-gamma and TNF-alpha and shed by the activity of the disintegrin-like metalloproteinase ADAM10

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283-293

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scholarly article

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10.2353/AJPATH.2009.080807

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1

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175

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2009-06-18T00:00:00Z

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26306702

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19541935

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multiple sclerosis

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2708814

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