Itch expression by Treg cells controls Th2 inflammatory responses.
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Ubiquitin-dependent regulation of Foxp3 and Treg functionTreg functional stability and its responsiveness to the microenvironmentSubsets of regulatory T cells and their roles in allergyMechanism-Based Strategies for the Management of Autoimmunity and Immune Dysregulation in Primary ImmunodeficienciesRegulatory T Cells Control Th2-Dominant Murine Autoimmune GastritisUpdate in asthma 2013Interleukin 4 promotes the development of ex-Foxp3 Th2 cells during immunity to intestinal helminthsNdfip1 restricts mTORC1 signalling and glycolysis in regulatory T cells to prevent autoinflammatory disease.Ubiquitous points of control over regulatory T cells.The E3 ubiquitin ligase Itch is required for the differentiation of follicular helper T cells.Regulation of T cell receptor complex-mediated signaling by ubiquitin and ubiquitin-like modificationsDeltex1 antagonizes HIF-1α and sustains the stability of regulatory T cells in vivo.Helicobacter pylori neutrophil-activating protein: a potential Treg modulator suppressing allergic asthma?Targeting Treg signaling for the treatment of autoimmune diseasesCytokine expression by invariant natural killer T cells is tightly regulated throughout development and settings of type-2 inflammation.Regulatory T cells as immunotherapy.The E3 ligase Itch in immune regulation and beyond.Regulatory T cell Itch reins in Th2 inflammation.GABAA receptor α4-subunit knockout enhances lung inflammation and airway reactivity in a murine asthma model.Ubiquitin Ligases and Deubiquitinating Enzymes in CD4+ T Cell Effector Fate Choice and Function.Fuelling the mechanisms of asthma: Increased fatty acid oxidation in inflammatory immune cells may represent a novel therapeutic target.An IκB Kinase-Regulated Feedforward Circuit Prolongs Inflammation.Regulatory T cells use "Itch" to control asthma.Pak2 is essential for the function of Foxp3+ regulatory T cells through maintaining a suppressive Treg phenotype.Immune Regulation by Ubiquitin Tagging as Checkpoint Code.Itch inhibits IL-17-mediated colon inflammation and tumorigenesis by ROR-γt ubiquitination.The ubiquitin ligase ITCH coordinates small intestinal epithelial homeostasis by modulating cell proliferation, differentiation, and migration.Astragaloside IV attenuates inflammatory reaction via activating immune function of regulatory T-cells inhibited by HMGB1 in mice.The E3 ubiquitin ligase Itch inhibits p38α signaling and skin inflammation through the ubiquitylation of Tab1.It takes CK2 to suppress TH2.
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Itch expression by Treg cells controls Th2 inflammatory responses.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 25 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Itch expression by Treg cells controls Th2 inflammatory responses.
@en
Itch expression by Treg cells controls Th2 inflammatory responses.
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type
label
Itch expression by Treg cells controls Th2 inflammatory responses.
@en
Itch expression by Treg cells controls Th2 inflammatory responses.
@nl
prefLabel
Itch expression by Treg cells controls Th2 inflammatory responses.
@en
Itch expression by Treg cells controls Th2 inflammatory responses.
@nl
P2093
P2860
P356
P1476
Itch expression by Treg cells controls Th2 inflammatory responses
@en
P2093
Chris Elly
Hyung-seung Jin
P2860
P304
P356
10.1172/JCI69355
P407
P50
P577
2013-10-25T00:00:00Z