Fibrinogen binding to ICAM-1 promotes EGFR-dependent mucin production in human airway epithelial cells.
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Fibrinogen β-derived Bβ(15-42) peptide protects against kidney ischemia/reperfusion injuryPharmacological and genetic depletion of fibrinogen protects from kidney fibrosis.Fibrinogen Is at the Interface of Host Defense and Pathogen Virulence in Staphylococcus aureus Infection.Inhibition of E-cadherin dependent cell-cell contact promotes MUC5AC mucin production through the activation of epidermal growth factor receptors.Comparison of comorbidities of migraine and tension headache in a pediatric headache clinic.TRPV1 and TRPA1 stimulation induces MUC5B secretion in the human nasal airway in vivo.
P2860
Fibrinogen binding to ICAM-1 promotes EGFR-dependent mucin production in human airway epithelial cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 08 May 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Fibrinogen binding to ICAM-1 p ...... human airway epithelial cells.
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Fibrinogen binding to ICAM-1 p ...... human airway epithelial cells.
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type
label
Fibrinogen binding to ICAM-1 p ...... human airway epithelial cells.
@en
Fibrinogen binding to ICAM-1 p ...... human airway epithelial cells.
@nl
prefLabel
Fibrinogen binding to ICAM-1 p ...... human airway epithelial cells.
@en
Fibrinogen binding to ICAM-1 p ...... human airway epithelial cells.
@nl
P2860
P1476
Fibrinogen binding to ICAM-1 p ...... human airway epithelial cells.
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P2093
P2860
P304
P356
10.1152/AJPLUNG.00032.2009
P577
2009-05-08T00:00:00Z