Prenatal hypoxia causes a sex-dependent increase in heart susceptibility to ischemia and reperfusion injury in adult male offspring: role of protein kinase C epsilon.
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Role of endothelin in uteroplacental circulation and fetal vascular functionSex-based differences in cardiac ischaemic injury and protection: therapeutic implicationsEarly origins of heart disease: low birth weight and determinants of cardiomyocyte endowment[Prenatal sex-specific programming and chronic diseases or Finis Ab Orígine Pendet].Antenatal hypoxia induces programming of reduced arterial blood pressure response in female rat offspring: role of ovarian function.Repeated exposure to methamphetamine induces sex-dependent hypersensitivity to ischemic injury in the adult rat heart.Prenatal Hypoxia Reduces Mitochondrial Protein Levels and Cytochrome c Oxidase Activity in Offspring Guinea Pig HeartsChronic prenatal hypoxia induces epigenetic programming of PKC{epsilon} gene repression in rat hearts.Developmental programming of cardiovascular dysfunction by prenatal hypoxia and oxidative stressFoetal nicotine exposure causes PKCε gene repression by promoter methylation in rat hearts.Foetal hypoxia increases cardiac AT(2)R expression and subsequent vulnerability to adult ischaemic injury.Hypoxia and fetal heart development.Prenatal exposure to hypoxia induced Beclin 1 signaling-mediated renal autophagy and altered renal development in rat fetusesMaternal hypoxia alters matrix metalloproteinase expression patterns and causes cardiac remodeling in fetal and neonatal ratsEpigenetic mechanisms in developmental programming of adult disease.Fetal hypoxia and programming of matrix metalloproteinases.Hypoxia-derived oxidative stress mediates epigenetic repression of PKCε gene in foetal rat hearts.Protective Effect of Antenatal Antioxidant on Nicotine-Induced Heart Ischemia-Sensitive Phenotype in Rat OffspringNormal lactational environment restores cardiomyocyte number after uteroplacental insufficiency: implications for the preterm neonateNorepinephrine causes epigenetic repression of PKCε gene in rodent hearts by activating Nox1-dependent reactive oxygen species production.Impact of oxidative stress in fetal programming.Estrogen Regulates Angiotensin II Receptor Expression Patterns and Protects the Heart from Ischemic Injury in Female Rats.Role of the hypothalamic-pituitary-adrenal axis in developmental programming of health and disease.Hypoxia inhibits cardiomyocyte proliferation in fetal rat hearts via upregulating TIMP-4Promoter methylation of Egr-1 site contributes to fetal hypoxia-mediated PKCε gene repression in the developing heartPostnatal resveratrol supplementation improves cardiovascular function in male and female intrauterine growth restricted offspringUmbilical artery systolic to diastolic ratio is associated with growth and myocardial performance in infants with hypoplastic left heart syndrome.Maternal obesity and the developmental programming of hypertension: a role for leptin.Developmental determinants of cardiac sensitivity to hypoxia.Developmental programming of cardiovascular disease by prenatal hypoxia.Preserved heart function after left ventricular pressure overload in adult mice subjected to neonatal cardiac hypoplasia.Ischemia/Reperfusion.Prenatal methamphetamine differentially alters myocardial sensitivity to ischemic injury in male and female adult hearts.Maternal High-Fat Diet Causes a Sex-Dependent Increase in AGTR2 Expression and Cardiac Dysfunction in Adult Male Rat Offspring.Chronic hypoxia increases peroxynitrite, MMP9 expression, and collagen accumulation in fetal guinea pig hearts.High altitude hypoxia and blood pressure dysregulation in adult chickens.MicroRNA-210 suppresses glucocorticoid receptor expression in response to hypoxia in fetal rat cardiomyocytes.Adverse Intrauterine Environment and Cardiac miRNA Expression.Impact of maternal cigarette smoke exposure on brain and kidney health outcomes in female offspring.Cardiac Development and Transcription Factors: Insulin Signalling, Insulin Resistance, and Intrauterine Nutritional Programming of Cardiovascular Disease.
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Prenatal hypoxia causes a sex-dependent increase in heart susceptibility to ischemia and reperfusion injury in adult male offspring: role of protein kinase C epsilon.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 26 May 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Prenatal hypoxia causes a sex- ...... e of protein kinase C epsilon.
@en
Prenatal hypoxia causes a sex- ...... e of protein kinase C epsilon.
@nl
type
label
Prenatal hypoxia causes a sex- ...... e of protein kinase C epsilon.
@en
Prenatal hypoxia causes a sex- ...... e of protein kinase C epsilon.
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prefLabel
Prenatal hypoxia causes a sex- ...... e of protein kinase C epsilon.
@en
Prenatal hypoxia causes a sex- ...... e of protein kinase C epsilon.
@nl
P2860
P356
P1476
Prenatal hypoxia causes a sex- ...... e of protein kinase C epsilon.
@en
P2093
P2860
P304
P356
10.1124/JPET.109.153239
P407
P577
2009-05-26T00:00:00Z