Quantitative immunohistochemical expression of c Kit in breast carcinomas is predictive of patients' outcome.
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Sunitinib significantly suppresses the proliferation, migration, apoptosis resistance, tumor angiogenesis and growth of triple-negative breast cancers but increases breast cancer stem cells.Nanotechniques Inactivate Cancer Stem Cells.Disparate impact of butyroyloxymethyl diethylphosphate (AN-7), a histone deacetylase inhibitor, and doxorubicin in mice bearing a mammary tumor.Soluble KIT correlates with clinical outcome in patients with metastatic breast cancer treated with sunitinibThe relevance of the intrinsic subtype to the clinicopathological features and biomarkers in Japanese breast cancer patients.Estrogen induces c-Kit and an aggressive phenotype in a model of invasive lobular breast cancer.FGF9 prevents pleural fibrosis induced by intrapleural adenovirus injection in mice.C-Kit receptor and tryptase expressing mast cells correlate with angiogenesis in breast cancer patients.Biofunctional characteristics of in situ and invasive breast carcinoma.
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Quantitative immunohistochemical expression of c Kit in breast carcinomas is predictive of patients' outcome.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 09 June 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Quantitative immunohistochemic ...... edictive of patients' outcome.
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Quantitative immunohistochemic ...... edictive of patients' outcome.
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type
label
Quantitative immunohistochemic ...... edictive of patients' outcome.
@en
Quantitative immunohistochemic ...... edictive of patients' outcome.
@nl
prefLabel
Quantitative immunohistochemic ...... edictive of patients' outcome.
@en
Quantitative immunohistochemic ...... edictive of patients' outcome.
@nl
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P1476
Quantitative immunohistochemic ...... edictive of patients' outcome.
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M-N Lavaut
S Carpentier
S Giusiano
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P2888
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10.1038/SJ.BJC.6605113
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P577
2009-06-09T00:00:00Z