Volatile anesthetic effects on midbrain-elicited locomotion suggest that the locomotor network in the ventral spinal cord is the primary site for immobility
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[What do we know about anesthetic mechanisms?: hypnosis, unresponsiveness to surgical incision and amnesia].Inhaled anesthetic responses of recombinant receptors and knockin mice harboring α2(S270H/L277A) GABA(A) receptor subunits that are resistant to isofluraneBrainstem regions affecting minimum alveolar concentration and movement pattern during isoflurane anesthesia.Altered anesthetic sensitivity of mice lacking Ndufs4, a subunit of mitochondrial complex I.Opposing actions of sevoflurane on GABAergic and glycinergic synaptic inhibition in the spinal ventral hornNitrous oxide-induced analgesia does not influence nitrous oxide's immobilizing requirements.Anesthetic effects on fictive locomotion in the rat isolated spinal cordValidation and insights of anesthetic action in an early vertebrate network: the isolated lamprey spinal cord.Identification and characterization of anesthetic targets by mouse molecular genetics approaches.Isoflurane inhibits synaptic vesicle exocytosis through reduced Ca2+ influx, not Ca2+-exocytosis coupling.Isoflurane, but Not the Nonimmobilizers F6 and F8, Inhibits Rat Spinal Cord Motor Neuron CaV1 Calcium Currents.The effect of nitrous oxide on the minimum alveolar concentration (MAC) and MAC derivatives of isoflurane in dogsNetwork actions of pentobarbital in the rat mesopontine tegmentum on sensory inflow through the spinothalamic tract.Sparing effect of robenacoxib on the minimum alveolar concentration for blunting adrenergic response (MAC-BAR) of sevoflurane in dogs.Interaction between maropitant and carprofen on sparing of the minimum alveolar concentration for blunting adrenergic response (MAC-BAR) of sevoflurane in dogs.Bidirectional modulation of isoflurane potency by intrathecal tetrodotoxin and veratridine in rats.Rat dorsal horn nociceptive-specific neurons are more sensitive than wide dynamic range neurons to depression by immobilizing doses of volatile anesthetics: an effect partially reversed by the opioid receptor antagonist naloxonePropofol produces immobility via action in the ventral horn of the spinal cord by a GABAergic mechanism.Regional knockdown of NDUFS4 implicates a thalamocortical circuit mediating anesthetic sensitivity.Electromyographic activation reveals cortical and sub-cortical dissociation during emergence from general anesthesia.
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Volatile anesthetic effects on midbrain-elicited locomotion suggest that the locomotor network in the ventral spinal cord is the primary site for immobility
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on June 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Volatile anesthetic effects on ...... he primary site for immobility
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Volatile anesthetic effects on ...... e primary site for immobility.
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type
label
Volatile anesthetic effects on ...... he primary site for immobility
@en
Volatile anesthetic effects on ...... e primary site for immobility.
@nl
prefLabel
Volatile anesthetic effects on ...... he primary site for immobility
@en
Volatile anesthetic effects on ...... e primary site for immobility.
@nl
P2093
P2860
P1433
P1476
Volatile anesthetic effects on ...... he primary site for immobility
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P2093
Milo Bravo
Shawn G Hayes
Steven L Jinks
P2860
P304
P356
10.1097/ALN.0B013E3181730297
P407
P577
2008-06-01T00:00:00Z