Repression of NF-kappaB and activation of AP-1 enhance apoptosis in prostate cancer cells. - Wikidata - wikimedia - TriplyDB

Repression of NF-kappaB and activation of AP-1 enhance apoptosis in prostate cancer cells.

Repression of NF-kappaB and activation of AP-1 enhance apoptosis in prostate cancer cells.

http://www.wikidata.org/entity/Q37275591

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Identification by automated screening of a small molecule that selectively eliminates neural stem cells derived from hESCs but not dopamine neurons beta-TrCP inhibition reduces prostate cancer cell growth via upregulation of the aryl hydrocarbon receptor.F-box protein 10, an NF-κB-dependent anti-apoptotic protein, regulates TRAIL-induced apoptosis through modulating c-Fos/c-FLIP pathway Activation of NF-{kappa}B by TMPRSS2/ERG Fusion Isoforms through Toll-Like Receptor-4.JunD-mediated repression of GADD45α and γ regulates escape from cell death in prostate cancer Inhibitor of Differentiation/DNA Binding 1 (ID1) Inhibits Etoposide-induced Apoptosis in a c-Jun/c-Fos-dependent Manner.Cytokine effects on cell viability and death of prostate carcinoma cells.Polyubiquitination of transforming growth factor β (TGFβ)-associated kinase 1 mediates nuclear factor-κB activation in response to different inflammatory stimuli.C-fos upregulates P-glycoprotein, contributing to the development of multidrug resistance in HEp-2 laryngeal cancer cells with VCR-induced resistance.

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Repression of NF-kappaB and activation of AP-1 enhance apoptosis in prostate cancer cells.

http://www.wikidata.org/entity/Q37275591

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on April 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Repression of NF-kappaB and ac ...... osis in prostate cancer cells.

@en

Repression of NF-kappaB and ac ...... osis in prostate cancer cells.

@nl

type

label

Repression of NF-kappaB and ac ...... osis in prostate cancer cells.

@en

Repression of NF-kappaB and ac ...... osis in prostate cancer cells.

@nl

prefLabel

Repression of NF-kappaB and ac ...... osis in prostate cancer cells.

@en

Repression of NF-kappaB and ac ...... osis in prostate cancer cells.

@nl

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International Journal of Cancer

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Repression of NF-kappaB and ac ...... osis in prostate cancer cells.

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Aria F Olumi

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Xiaoping Zhang

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Xu Huang

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P2860

Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

Inhibition of death receptor signals by cellular FLIP

TRAIL receptor-2 signals apoptosis through FADD and caspase-8

Cellular FLICE-inhibitory protein splice variants inhibit different steps of caspase-8 activation at the CD95 death-inducing signaling complex

A role for AP-1 in apoptosis: the case for and against

An antagonist decoy receptor and a death domain-containing receptor for TRAIL

Increased insulin-like growth factor I receptor expression and signaling are components of androgen-independent progression in a lineage-derived prostate cancer progression model

Constitutive activation of IkappaB kinase alpha and NF-kappaB in prostate cancer cells is inhibited by ibuprofen

The E3 ubiquitin ligase itch couples JNK activation to TNFalpha-induced cell death by inducing c-FLIP(L) turnover

FADD/MORT1 and caspase-8 are recruited to TRAIL receptors 1 and 2 and are essential for apoptosis mediated by TRAIL receptor 2

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1980-1989

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10.1002/IJC.24139

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8

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124

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19123467

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apoptotic process

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