Induction of cytoplasmic accumulation of p53: a mechanism for low levels of arsenic exposure to predispose cells for malignant transformation.
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mot-2-Mediated cross talk between nuclear factor-B and p53 is involved in arsenite-induced tumorigenesis of human embryo lung fibroblast cellsA MALAT1/HIF-2α feedback loop contributes to arsenite carcinogenesisMolecular features in arsenic-induced lung tumorsOncogenomic disruptions in arsenic-induced carcinogenesis.Arsenite induced poly(ADP-ribosyl)ation of tumor suppressor P53 in human skin keratinocytes as a possible mechanism for carcinogenesis associated with arsenic exposure.The effects of arsenic trioxide on DNA synthesis and genotoxicity in human colon cancer cellsInfluence of arsenate and arsenite on signal transduction pathways: an updateFocal adhesion kinase and p53 synergistically decrease neuroblastoma cell survival.Low-dose arsenic induces chemotherapy protection via p53/NF-κB-mediated metabolic regulation.p53-Based Strategy for Protection of Bone Marrow From Y-90 Ibritumomab Tiuxetan.Polycomb (PcG) proteins, BMI1 and SUZ12, regulate arsenic-induced cell transformationEffects of arsenic exposure on DNA methylation in cord blood samples from newborn babies and in a human lymphoblast cell lineArsenite Regulates Prolongation of Glycan Residues of Membrane Glycoprotein: A Pivotal Study via Wax Physisorption Kinetics and FTIR Imaging.Genetic and epigenetic effects of environmental arsenicals.Low-dose radiation exposure induces a HIF-1-mediated adaptive and protective metabolic response.Low-dose arsenic-mediated metabolic shift is associated with activation of Polo-like kinase 1 (Plk1).Molecular changes during arsenic-induced cell transformation.Activation of the p38 MAPK/Akt/ERK1/2 signal pathways is required for the protein stabilization of CDC6 and cyclin D1 in low-dose arsenite-induced cell proliferation.The repressive effect of NF-kappaB on p53 by mot-2 is involved in human keratinocyte transformation induced by low levels of arsenite.Induction of neoplastic transformation by ectopic expression of human aldo-keto reductase 1C isoforms in NIH3T3 cells.Low dose arsenite confers resistance to UV induced apoptosis via p53-MDM2 pathway in ketatinocytes.Arsenic trioxide targets MTHFD1 and SUMO-dependent nuclear de novo thymidylate biosynthesis.Differentially Expressed mRNA Targets of Differentially Expressed miRNAs Predict Changes in The TP53 Axis and Carcinogenesis Related Pathways in Human Keratinocytes Chronically Exposed to Arsenic.Arsenic-Induced Carcinogenesis: The Impact of miRNA DysregulationUse of Arsenic-Induced Palmoplantar Hyperkeratosis and Skin Cancers to Predict Risk of Subsequent Internal Malignancy
P2860
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P2860
Induction of cytoplasmic accumulation of p53: a mechanism for low levels of arsenic exposure to predispose cells for malignant transformation.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Induction of cytoplasmic accum ...... for malignant transformation.
@en
Induction of cytoplasmic accum ...... for malignant transformation.
@nl
type
label
Induction of cytoplasmic accum ...... for malignant transformation.
@en
Induction of cytoplasmic accum ...... for malignant transformation.
@nl
prefLabel
Induction of cytoplasmic accum ...... for malignant transformation.
@en
Induction of cytoplasmic accum ...... for malignant transformation.
@nl
P2093
P2860
P50
P1433
P1476
Induction of cytoplasmic accum ...... s for malignant transformation
@en
P2093
Jianglin Zhang
Kevin T McHenry
Mihee M Kim
Vanessa Lopez-Pajares
Zhi-Min Yuan
P2860
P304
P356
10.1158/0008-5472.CAN-08-3025
P407
P577
2008-11-01T00:00:00Z