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Sudden infant death syndrome: do ion channels play a role?

Sudden infant death syndrome: do ion channels play a role?

http://www.wikidata.org/entity/Q37281775

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Sodium channel β subunits: emerging targets in channelopathies A "Wear and Tear" Hypothesis to Explain Sudden Infant Death Syndrome.Genomic risk factors in sudden infant death syndrome Cardiac ion channelopathies and the sudden infant death syndrome Sudden infant death syndrome in Korea: a retrospective analysis of autopsy-diagnosed cases Long QT syndrome-associated mutations in intrauterine fetal death.Cardiac channelopathies and sudden infant death syndrome.Sudden infant death syndrome caused by cardiac arrhythmias: only a matter of genes encoding ion channels?Sudden unexpected death in infancy in Denmark.Voltage-Gated Sodium Channel β Subunits and Their Related Diseases.Comparative proteome analysis for identification of differentially abundant proteins in SIDS.

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Sudden infant death syndrome: do ion channels play a role?

http://www.wikidata.org/entity/Q37281775

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 31 July 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Sudden infant death syndrome: do ion channels play a role?

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Sudden infant death syndrome: do ion channels play a role?

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Sudden infant death syndrome: do ion channels play a role?

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Sudden infant death syndrome: do ion channels play a role?

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Sudden infant death syndrome: do ion channels play a role?

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Sudden infant death syndrome: do ion channels play a role?

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J.HRTHM.2008.07.028

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Sudden infant death syndrome: do ion channels play a role?

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David W Van Norstrand

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Michael J Ackerman

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P2860

MiRP1 forms IKr potassium channels with HERG and is associated with cardiac arrhythmia

Mutations in Kir2.1 cause the developmental and episodic electrical phenotypes of Andersen's syndrome

Ankyrin-B mutation causes type 4 long-QT cardiac arrhythmia and sudden cardiac death

Novel mechanism for sudden infant death syndrome: persistent late sodium current secondary to mutations in caveolin-3

Mutation in glycerol-3-phosphate dehydrogenase 1 like gene (GPD1-L) decreases cardiac Na+ current and causes inherited arrhythmias

Mutation of an A-kinase-anchoring protein causes long-QT syndrome

Syntrophin mutation associated with long QT syndrome through activation of the nNOS-SCN5A macromolecular complex

Mutant caveolin-3 induces persistent late sodium current and is associated with long-QT syndrome

Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias

SCN4B-encoded sodium channel beta4 subunit in congenital long-QT syndrome

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10.1016/J.HRTHM.2008.07.028

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