Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
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Microevolution of group A streptococci in vivo: capturing regulatory networks engaged in sociomicrobiology, niche adaptation, and hypervirulenceA genome-wide analysis of small regulatory RNAs in the human pathogen group A StreptococcusThe streptococcal collagen-like protein-1 (Scl1) is a significant determinant for biofilm formation by group a StreptococcusThe group A streptococcal collagen-like protein-1, Scl1, mediates biofilm formation by targeting the extra domain A-containing variant of cellular fibronectin expressed in wounded tissueMolecular epidemiology and genomics of group A StreptococcusStreptococcal M1 protein constructs a pathological host fibrinogen networkRole for streptococcal collagen-like protein 1 in M1T1 group A Streptococcus resistance to neutrophil extracellular trapsEvolutionary Constraints Shaping Streptococcus pyogenes-Host Interactions.Tracing the evolutionary history of the pandemic group A streptococcal M1T1 clone.Complete Genome Sequence of emm1 Streptococcus pyogenes A20, a Strain with an Intact Two-Component System, CovRS, Isolated from a Patient with Necrotizing Fasciitis.Coiled-coil irregularities of the M1 protein structure promote M1-fibrinogen interaction and influence group A Streptococcus host cell interactions and virulence.Clinical and microbial characteristics of invasive Streptococcus pyogenes disease in New Caledonia, a region in Oceania with a high incidence of acute rheumatic fever.Evolutionary pathway to increased virulence and epidemic group A Streptococcus disease derived from 3,615 genome sequences.Wiring the streptococcal network for alternative lifestylesStreptococcal collagen-like protein A and general stress protein 24 are immunomodulating virulence factors of group A Streptococcus.M1T1 group A streptococcal pili promote epithelial colonization but diminish systemic virulence through neutrophil extracellular entrapment.Genetic switch to hypervirulence reduces colonization phenotypes of the globally disseminated group A streptococcus M1T1 clone.Study of the IgG endoglycosidase EndoS in group A streptococcal phagocyte resistance and virulence.Signal transduction through CsrRS confers an invasive phenotype in group A Streptococcus.M protein and hyaluronic acid capsule are essential for in vivo selection of covRS mutations characteristic of invasive serotype M1T1 group A Streptococcus.Streptolysin O and NAD-glycohydrolase prevent phagolysosome acidification and promote group A Streptococcus survival in macrophages.The contribution of group A streptococcal virulence determinants to the pathogenesis of sepsis.Disease manifestations and pathogenic mechanisms of Group A Streptococcus.Retargeting pre-existing human antibodies to a bacterial pathogen with an alpha-Gal conjugated aptamerAntibacterial activity of the contact and complement systems is blocked by SIC, a protein secreted by Streptococcus pyogenesUnique genomic arrangements in an invasive serotype M23 strain of Streptococcus pyogenes identify genes that induce hypervirulence.Partial loss of CovS function in Streptococcus pyogenes causes severe invasive diseaseTranscriptional regulation of a bacteriophage encoded extracellular DNase (Spd-3) by Rgg in Streptococcus pyogenes.A key role for the urokinase plasminogen activator (uPA) in invasive Group A streptococcal infection.Microarray analysis of microbiota of gingival lesions in noma patients.Counteractive balancing of transcriptome expression involving CodY and CovRS in Streptococcus pyogenes.Streptococcal inhibitor of complement promotes innate immune resistance phenotypes of invasive M1T1 group A StreptococcusParameters governing invasive disease propensity of non-M1 serotype group A streptococci.M1 protein allows Group A streptococcal survival in phagocyte extracellular traps through cathelicidin inhibition.Postpartum group a Streptococcus sepsis and maternal immunology.Zinc disrupts central carbon metabolism and capsule biosynthesis in Streptococcus pyogenes.A group A Streptococcus ADP-ribosyltransferase toxin stimulates a protective interleukin 1β-dependent macrophage immune responseSerine/threonine phosphatase (SP-STP), secreted from Streptococcus pyogenes, is a pro-apoptotic protein.Targeted Curing of All Lysogenic Bacteriophage from Streptococcus pyogenes Using a Novel Counter-selection TechniqueClinical and microbiologic characteristics of invasive Streptococcus pyogenes infections in north and south India.
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Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
@en
Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
@nl
type
label
Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
@en
Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
@nl
prefLabel
Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
@en
Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
@nl
P2860
P356
P1476
Rise and persistence of global M1T1 clone of Streptococcus pyogenes.
@en
P2093
Malak Kotb
P2860
P304
P356
10.3201/EID1410.071660
P50
P577
2008-10-01T00:00:00Z