Paracrine transactivation of the CB1 cannabinoid receptor by AT1 angiotensin and other Gq/11 protein-coupled receptors
about
International Union of Basic and Clinical Pharmacology. LXXIX. Cannabinoid receptors and their ligands: beyond CB₁ and CB₂Pharmacological characterization of GPR55, a putative cannabinoid receptorRegulation of endocannabinoid release by G proteins: a paracrine mechanism of G protein-coupled receptor action.Multiple functions of endocannabinoid signaling in the brain.Acute and chronic systemic CB1 cannabinoid receptor blockade improves blood pressure regulation and metabolic profile in hypertensive (mRen2)27 rats.Functional enhancement of AT1R potency in the presence of the TPαR is revealed by a comprehensive 7TM receptor co-expression screen.Overactive cannabinoid 1 receptor in podocytes drives type 2 diabetic nephropathy.Cannabinoid receptor expression in HIV encephalitis and HIV-associated neuropathologic comorbidities.Endocannabinoid tone versus constitutive activity of cannabinoid receptors.Angiotensin II induces vascular endocannabinoid release, which attenuates its vasoconstrictor effect via CB1 cannabinoid receptors.Fatty Acid modulation of the endocannabinoid system and the effect on food intake and metabolism.Medullary Endocannabinoids Contribute to the Differential Resting Baroreflex Sensitivity in Rats with Altered Brain Renin-Angiotensin System Expression.Clozapine, atypical antipsychotics, and the benefits of fast-off D2 dopamine receptor antagonism.CB1 receptor antagonists: new discoveries leading to new perspectives.Opposing local effects of endocannabinoids on the activity of noradrenergic neurons and release of noradrenaline: relevance for their role in depression and in the actions of CB(1) receptor antagonists.No more pain upon Gq-protein-coupled receptor activation: role of endocannabinoids.Heterologous regulation of the cannabinoid type 1 receptor by angiotensin II in astrocytes of spontaneously hypertensive rats.Endocannabinoids modulate Gq/11 protein-coupled receptor agonist-induced vasoconstriction via a negative feedback mechanism.Heterologous phosphorylation-induced formation of a stability lock permits regulation of inactive receptors by β-arrestins.Activation of CB1 receptors by 2-arachidonoylglycerol attenuates vasoconstriction induced by U46619 and angiotensin II in human and rat pulmonary arteries.
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P2860
Paracrine transactivation of the CB1 cannabinoid receptor by AT1 angiotensin and other Gq/11 protein-coupled receptors
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on 07 April 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Paracrine transactivation of t ...... q/11 protein-coupled receptors
@en
Paracrine transactivation of t ...... /11 protein-coupled receptors.
@nl
type
label
Paracrine transactivation of t ...... q/11 protein-coupled receptors
@en
Paracrine transactivation of t ...... /11 protein-coupled receptors.
@nl
prefLabel
Paracrine transactivation of t ...... q/11 protein-coupled receptors
@en
Paracrine transactivation of t ...... /11 protein-coupled receptors.
@nl
P2093
P2860
P921
P356
P1476
Paracrine transactivation of t ...... q/11 protein-coupled receptors
@en
P2093
George Kunos
György Bagdy
Gábor Turu
László Hunyady
László Offertaler
László Szidonya
Pál Gyombolai
Péter Várnai
P2860
P304
16914-16921
P356
10.1074/JBC.M109.003681
P407
P50
P577
2009-04-07T00:00:00Z