Transforming growth factor-beta promotes recruitment of bone marrow cells and bone marrow-derived mesenchymal stem cells through stimulation of MCP-1 production in vascular smooth muscle cells
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TGF-β/Smad3 stimulates stem cell/developmental gene expression and vascular smooth muscle cell de-differentiationStem Cells and Regenerative Medicine: Myth or Reality of the 21th CenturyHypoxia is essential for bone-tendon junction healing: the molecular biological evidence.Role of the TGF-β/BMP-7/Smad pathways in renal diseasesAging mesenchymal stem cells fail to protect because of impaired migration and antiinflammatory response.Protein kinase C delta mediates arterial injury responses through regulation of vascular smooth muscle cell apoptosis.MCP/CCR2 signaling is essential for recruitment of mesenchymal progenitor cells during the early phase of fracture healing.Advanced glycation end products induce chemokine/cytokine production via activation of p38 pathway and inhibit proliferation and migration of bone marrow mesenchymal stem cells.Skeletal trauma generates systemic BMP2 activation that is temporally related to the mobilization of CD73+ cells.TGF-β and restenosis revisited: a Smad link.Fibroblast activation protein (FAP) is essential for the migration of bone marrow mesenchymal stem cells through RhoA activationTissue engineering bone using autologous progenitor cells in the peritoneum.Use of human mesenchymal stem cells as alternative source of smooth muscle cells in vessel engineeringMyocardial production and release of MCP-1 and SDF-1 following myocardial infarction: differences between mice and man.Identification of chemoattractive factors involved in the migration of bone marrow-derived mesenchymal stem cells to brain lesions caused by prionsProtein kinase C-delta mediates adventitial cell migration through regulation of monocyte chemoattractant protein-1 expression in a rat angioplasty modelTGF-β and Smad3 modulate PI3K/Akt signaling pathway in vascular smooth muscle cellsIn vivo tracking of mesechymal stem cells using fluorescent nanoparticles in an osteochondral repair model.The expression of P2X₇ receptors in EPCs and their potential role in the targeting of EPCs to brain gliomasProtein kinase N1 is a novel substrate of NFATc1-mediated cyclin D1-CDK6 activity and modulates vascular smooth muscle cell division and migration leading to inward blood vessel wall remodelingInjury-activated transforming growth factor β controls mobilization of mesenchymal stem cells for tissue remodeling.Reduction of intimal hyperplasia in injured rat arteries promoted by catheter balloons coated with polyelectrolyte multilayers that contain plasmid DNA encoding PKCδ.Ligand-dependent actions of the vitamin D receptor are required for activation of TGF-β signaling during the inflammatory response to cutaneous injuryTGF-β mediates homing of bone marrow-derived human mesenchymal stem cells to glioma stem cellsGene expression profiles of human adipose tissue-derived mesenchymal stem cells are modified by cell culture density.Protein kinase C-δ (PKCδ) regulates proinflammatory chemokine expression through cytosolic interaction with the NF-κB subunit p65 in vascular smooth muscle cells.Smooth muscle cell-specific Tgfbr1 deficiency attenuates neointimal hyperplasia but promotes an undesired vascular phenotype for injured arteries.Mechanism of arterial remodeling in chronic allograft vasculopathy.The therapeutic potential of bone marrow-derived mesenchymal stromal cells on hepatocellular carcinoma.Biomaterial implants mediate autologous stem cell recruitment in mice.Stem cells and vascular regenerative medicine: A mini review.Inflammatory Chemokines MIP-1δ and MIP-3α Are Involved in the Migration of Multipotent Mesenchymal Stromal Cells Induced by Hepatoma Cells.Tissue Engineering of Cartilage; Can Cannabinoids Help?Platelet-rich concentrates differentially release growth factors and induce cell migration in vitro.The novel function of advanced glycation end products in regulation of MMP-9 production.Biological characteristics of foam cell formation in smooth muscle cells derived from bone marrow stem cells.Transforming growth factor-β increases vascular smooth muscle cell proliferation through the Smad3 and extracellular signal-regulated kinase mitogen-activated protein kinases pathways.TGF-beta in the pathogenesis and prevention of disease: a matter of aneurysmic proportions.TGFβ3 recruits endogenous mesenchymal stem cells to initiate bone regeneration.Monozygotic twins discordant for recessive dystrophic epidermolysis bullosa phenotype highlight the role of TGF-β signalling in modifying disease severity.
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P2860
Transforming growth factor-beta promotes recruitment of bone marrow cells and bone marrow-derived mesenchymal stem cells through stimulation of MCP-1 production in vascular smooth muscle cells
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 30 April 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Transforming growth factor-bet ...... n vascular smooth muscle cells
@en
Transforming growth factor-bet ...... vascular smooth muscle cells.
@nl
type
label
Transforming growth factor-bet ...... n vascular smooth muscle cells
@en
Transforming growth factor-bet ...... vascular smooth muscle cells.
@nl
prefLabel
Transforming growth factor-bet ...... n vascular smooth muscle cells
@en
Transforming growth factor-bet ...... vascular smooth muscle cells.
@nl
P2093
P2860
P356
P1476
Transforming growth factor-bet ...... n vascular smooth muscle cells
@en
P2093
Chunjie Wang
Dai Yamanouchi
K Craig Kent
Kaori Kato
Shahin Rafii
Shirling Tsai
P2860
P304
17564-17574
P356
10.1074/JBC.M109.013987
P407
P577
2009-04-30T00:00:00Z