Phosphodiesterase 5 inhibition blocks pressure overload-induced cardiac hypertrophy independent of the calcineurin pathway.
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Left Ventricular Hypertrophy in Chronic Kidney Disease Patients: From Pathophysiology to Treatment.The immediate effect of sildenafil on right ventricular function in patients with heart failure measured by cardiac magnetic resonance: a randomized control trialSildenafil ameliorates left ventricular T-tubule remodeling in a pressure overload-induced murine heart failure modelCyclic GMP/PKG-dependent inhibition of TRPC6 channel activity and expression negatively regulates cardiomyocyte NFAT activation Novel mechanism of cardiac stress modulation by PDE5 inhibitionVolume overload induces differential spatiotemporal regulation of myocardial soluble guanylyl cyclase in eccentric hypertrophy and heart failurePhosphorylation of TRPC6 channels at Thr69 is required for anti-hypertrophic effects of phosphodiesterase 5 inhibitioncGMP-dependent protein kinases and cGMP phosphodiesterases in nitric oxide and cGMP action.Right ventricular failure due to chronic pressure load: What have we learned in animal models since the NIH working group statement?Right ventricular cyclic nucleotide signaling is decreased in hyperoxia-induced pulmonary hypertension in neonatal mice.Cardioprotective actions of cyclic GMP: lessons from genetic animal models.Gallic acid prevents isoproterenol-induced cardiac hypertrophy and fibrosis through regulation of JNK2 signaling and Smad3 binding activity.Chronic inhibition of cGMP-specific phosphodiesterase 5 suppresses endoplasmic reticulum stress in heart failure.Cyclic GMP signaling in cardiovascular pathophysiology and therapeutics.Phosphodiesterase 5 inhibitors: are they cardioprotective?The failing Fontan: etiology, diagnosis and management.Phosphodiesterases and cyclic GMP regulation in heart muscle.Moving into a new neighborhood: NOS goes nuclear.Effects of long-term atorvastatin treatment on cardiac aging.Chronic inhibition of cyclic guanosine monophosphate-specific phosphodiesterase 5 prevented cardiac fibrosis through inhibition of transforming growth factor β-induced Smad signaling.Vasostatin-1 Stops Structural Remodeling and Improves Calcium Handling via the eNOS-NO-PKG Pathway in Rat Hearts Subjected to Chronic β-Adrenergic Receptor Activation.The ability of phosphodiesterase-5 inhibitors sildenafil and ordonafil to reverse L-NAME induced cardiac hypertrophy in the rabbit: possible role of calcineurin and p38.
P2860
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P2860
Phosphodiesterase 5 inhibition blocks pressure overload-induced cardiac hypertrophy independent of the calcineurin pathway.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Phosphodiesterase 5 inhibition ...... nt of the calcineurin pathway.
@en
Phosphodiesterase 5 inhibition ...... nt of the calcineurin pathway.
@nl
type
label
Phosphodiesterase 5 inhibition ...... nt of the calcineurin pathway.
@en
Phosphodiesterase 5 inhibition ...... nt of the calcineurin pathway.
@nl
prefLabel
Phosphodiesterase 5 inhibition ...... nt of the calcineurin pathway.
@en
Phosphodiesterase 5 inhibition ...... nt of the calcineurin pathway.
@nl
P2093
P2860
P356
P1476
Phosphodiesterase 5 inhibition ...... nt of the calcineurin pathway.
@en
P2093
David A Kass
Djahida Bedja
Eiki Takimoto
Kathleen L Gabrielson
Manling Zhang
Norimichi Koitabashi
Steven Hsu
Takahiro Nagayama
P2860
P304
P356
10.1093/CVR/CVN324
P577
2008-11-24T00:00:00Z