A potent soluble epoxide hydrolase inhibitor, t-AUCB, acts through PPARγ to modulate the function of endothelial progenitor cells from patients with acute myocardial infarction

A potent soluble epoxide hydrolase inhibitor, t-AUCB, acts through PPARγ to modulate the function of endothelial progenitor cells from patients with acute myocardial infarction

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http://www.wikidata.org/entity/Q37291641
Epoxyeicosatrienoic acids and cardioprotection: the road to translationSoluble epoxide hydrolase: A potential target for metabolic diseasesSoluble epoxide hydrolase: gene structure, expression and deletionPPARγ signaling is required for mediating EETs protective effects in neonatal cardiomyocytes exposed to LPSHigh salt diet modulates vascular response in A2AAR (+/+) and A 2AAR (-/-) mice: role of sEH, PPARγ, and K ATP channels.Inhibition of soluble epoxide hydrolase increases coronary perfusion in miceRoles of endothelial nitric oxide synthase (eNOS) and mitochondrial permeability transition pore (MPTP) in epoxyeicosatrienoic acid (EET)-induced cardioprotection against infarction in intact rat hearts.Aerobic exercise-based rehabilitation affects the activities of progenitor endothelial cells through EETs pathway.The role of 14,15-dihydroxyeicosatrienoic acid levels in inflammation and its relationship to lipoproteins.Peroxisome proliferator-activated receptor γ (PPARγ) mediates the protective effect of quercetin against myocardial ischemia-reperfusion injury via suppressing the NF-κB pathway.Inhibition of soluble epoxide hydrolase augments astrocyte release of vascular endothelial growth factor and neuronal recovery after oxygen-glucose deprivation.TPPU enhanced exercise-induced epoxyeicosatrienoic acid concentrations to exert cardioprotection in mice after myocardial infarction.Metabolic/inflammatory/vascular comorbidity in psychiatric disorders; soluble epoxide hydrolase (sEH) as a possible new target.Peroxisome Proliferator-Activated Receptor Gamma Promotes Mesenchymal Stem Cells to Express Connexin43 via the Inhibition of TGF-β1/Smads Signaling in a Rat Model of Myocardial Infarction.PIAS1 protects against myocardial ischemia-reperfusion injury by stimulating PPARγ SUMOylation
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A potent soluble epoxide hydrolase inhibitor, t-AUCB, acts through PPARγ to modulate the function of endothelial progenitor cells from patients with acute myocardial infarction

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http://www.wikidata.org/entity/Q37291641
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on 21 April 2012
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
A potent soluble epoxide hydro ...... th acute myocardial infarction
@en
A potent soluble epoxide hydro ...... h acute myocardial infarction.
@nl
type
label
A potent soluble epoxide hydro ...... th acute myocardial infarction
@en
A potent soluble epoxide hydro ...... h acute myocardial infarction.
@nl
prefLabel
A potent soluble epoxide hydro ...... th acute myocardial infarction
@en
A potent soluble epoxide hydro ...... h acute myocardial infarction.
@nl
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A potent soluble epoxide hydro ...... th acute myocardial infarction
@en
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P2860
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P356
10.1016/J.IJCARD.2012.03.167
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