Acute hyperglycaemia enhances oxidative stress and aggravates myocardial ischaemia/reperfusion injury: role of thioredoxin-interacting protein. - Wikidata - wikimedia - TriplyDB

Acute hyperglycaemia enhances oxidative stress and aggravates myocardial ischaemia/reperfusion injury: role of thioredoxin-interacting protein.

Acute hyperglycaemia enhances oxidative stress and aggravates myocardial ischaemia/reperfusion injury: role of thioredoxin-interacting protein.

http://www.wikidata.org/entity/Q37295345

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Deletion of thioredoxin-interacting protein improves cardiac inotropic reserve in the streptozotocin-induced diabetic heart The Emerging Role of Thioredoxin-Interacting Protein in Myocardial Ischemia/Reperfusion Injury Thioredoxin-Interacting Protein Mediates Apoptosis in Early Brain Injury after Subarachnoid Haemorrhage Oxidative stress contributes to orthopedic trauma-induced acute kidney injury in obese rats.Influence of glycemic control on gain in VO2 peak, in patients with type 2 diabetes enrolled in cardiac rehabilitation after an acute coronary syndrome. The prospective DARE study Berberine Attenuates Myocardial Ischemia/Reperfusion Injury by Reducing Oxidative Stress and Inflammation Response: Role of Silent Information Regulator 1.Up-regulation of microRNA-135a protects against myocardial ischemia/reperfusion injury by decreasing TXNIP expression in diabetic mice.Berberine protects rat heart from ischemia/reperfusion injury via activating JAK2/STAT3 signaling and attenuating endoplasmic reticulum stress.Achyranthes bidentata polypeptides reduces oxidative stress and exerts protective effects against myocardial ischemic/reperfusion injury in rats.Inhibition of ALDH2 by O-GlcNAcylation contributes to the hyperglycemic exacerbation of myocardial ischemia/reperfusion injury.Targeting danger-associated molecular patterns after myocardial infarction.Dexmedetomidine protects against renal ischemia and reperfusion injury by inhibiting the P38-MAPK/TXNIP signaling activation in streptozotocin induced diabetic rats.Thioredoxin-Interacting Protein Mediates NLRP3 Inflammasome Activation Involved in the Susceptibility to Ischemic Acute Kidney Injury in Diabetes.Insights for Oxidative Stress and mTOR Signaling in Myocardial Ischemia/Reperfusion Injury under Diabetes.Myocyte membrane and microdomain modifications in diabetes: determinants of ischemic tolerance and cardioprotection.Protective Effects of Pterostilbene Against Myocardial Ischemia/Reperfusion Injury in Rats.Melatonin rescues cardiac thioredoxin system during ischemia-reperfusion injury in acute hyperglycemic state by restoring Notch1/Hes1/Akt signaling in a membrane receptor-dependent manner.SIRT1 activation by pterostilbene attenuates the skeletal muscle oxidative stress injury and mitochondrial dysfunction induced by ischemia reperfusion injury.Icaritin Attenuates Myocardial Ischemia and Reperfusion Injury Via Anti-Inflammatory and Anti-Oxidative Stress Effects in Rats.Apocynum venetum leaf attenuates myocardial ischemia/reperfusion injury by inhibiting oxidative stress.Melatonin receptor-mediated protection against myocardial ischemia/reperfusion injury: role of SIRT1.Tongxinluo attenuates reperfusion injury in diabetic hearts by angiopoietin-like 4-mediated protection of endothelial barrier integrity via PPAR-α pathway.

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Acute hyperglycaemia enhances oxidative stress and aggravates myocardial ischaemia/reperfusion injury: role of thioredoxin-interacting protein.

http://www.wikidata.org/entity/Q37295345

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article científic

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bilimsel makale

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scientific article published on 11 January 2013

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Acute hyperglycaemia enhances ...... ioredoxin-interacting protein.

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Acute hyperglycaemia enhances ...... ioredoxin-interacting protein.

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Acute hyperglycaemia enhances ...... ioredoxin-interacting protein.

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J.1582-4934.2012.01661.X

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Journal of Cellular and Molecular Medicine

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Acute hyperglycaemia enhances ...... ioredoxin-interacting protein.

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Feng Gao

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Haifeng Zhang

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Heping Zhou

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Hui Su

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Lele Ji

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Wei Zhang

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Wenjuan Xing

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Xiaoming Wang

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Xin Sun

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Xinhong Qian

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P2860

Myocardial ischemia/reperfusion-injury, a clinical view on a complex pathophysiological process

Hyperglycemia promotes oxidative stress through inhibition of thioredoxin function by thioredoxin-interacting protein

Vitamin D3 up-regulated protein 1 mediates oxidative stress via suppressing the thioredoxin function

Myocardial infarction in diabetic rats: role of hyperglycaemia on infarct size and early expression of hypoxia-inducible factor 1

Vitamin D(3)-up-regulated protein-1 is a stress-responsive gene that regulates cardiomyocyte viability through interaction with thioredoxin

p38 mitogen-activated protein kinase: a critical node linking insulin resistance and cardiovascular diseases in type 2 diabetes mellitus.

Nitric oxide-dependent suppression of thioredoxin-interacting protein expression enhances thioredoxin activity.

TXNIP regulates peripheral glucose metabolism in humans.

Intracellular shuttling and mitochondrial function of thioredoxin-interacting protein.

Stress hyperglycaemia and increased risk of death after myocardial infarction in patients with and without diabetes: a systematic overview.

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10.1111/J.1582-4934.2012.01661.X

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reperfusion injury

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