REST reduction is essential for hypoxia-induced neuroendocrine differentiation of prostate cancer cells by activating autophagy signaling. - Wikidata - wikimedia - TriplyDB

REST reduction is essential for hypoxia-induced neuroendocrine differentiation of prostate cancer cells by activating autophagy signaling.

REST reduction is essential for hypoxia-induced neuroendocrine differentiation of prostate cancer cells by activating autophagy signaling.

http://www.wikidata.org/entity/Q37295602

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REST is a hypoxia-responsive transcriptional repressor MAOA-a novel decision maker of apoptosis and autophagy in hormone refractory neuroendocrine prostate cancer cells.REST is a crucial regulator for acquiring EMT-like and stemness phenotypes in hormone-refractory prostate cancer.Establishment of a neuroendocrine prostate cancer model driven by the RNA splicing factor SRRM4.Andrographolide sensitizes prostate cancer cells to TRAIL-induced apoptosis.Implications of PI3K/AKT inhibition on REST protein stability and neuroendocrine phenotype acquisition in prostate cancer cells.Histone demethylase PHF8 regulates hypoxia signaling through HIF1α and H3K4me3.Development of Neuroendocrine Prostate Cancers by the Ser/Arg Repetitive Matrix 4-Mediated RNA Splicing Network.TNFAIP8 promotes prostate cancer cell survival by inducing autophagy.Hypoxia and Chromatin: A Focus on Transcriptional Repression Mechanisms.A novel mechanism of SRRM4 in promoting neuroendocrine prostate cancer development via a pluripotency gene network

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REST reduction is essential for hypoxia-induced neuroendocrine differentiation of prostate cancer cells by activating autophagy signaling.

http://www.wikidata.org/entity/Q37295602

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 28 March 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

REST reduction is essential fo ...... ctivating autophagy signaling.

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REST reduction is essential fo ...... ctivating autophagy signaling.

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REST reduction is essential fo ...... ctivating autophagy signaling.

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REST reduction is essential fo ...... ctivating autophagy signaling.

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REST reduction is essential fo ...... ctivating autophagy signaling.

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REST reduction is essential fo ...... ctivating autophagy signaling.

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ONCOTARGET.8433

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REST reduction is essential fo ...... ctivating autophagy signaling.

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Allen W Chiu

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Cheng-Ying Chu

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Chi-Hung Lin

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Chia-Yang Cheng

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Chin-Chen Pan

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Hsiao-Jen Chung

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Hsing-Jien Kung

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Mel Campbell

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Pei-Ching Chang

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Shu-Huei Shen

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P2860

CDYL bridges REST and histone methyltransferases for gene repression and suppression of cellular transformation

Siah2-dependent concerted activity of HIF and FoxA2 regulates formation of neuroendocrine phenotype and neuroendocrine prostate tumors

SCFbeta-TRCP controls oncogenic transformation and neural differentiation through REST degradation

Interleukin-6- and cyclic AMP-mediated signaling potentiates neuroendocrine differentiation of LNCaP prostate tumor cells.

Hypoxia-induced autophagy is mediated through hypoxia-inducible factor induction of BNIP3 and BNIP3L via their BH3 domains

A genetic screen for candidate tumor suppressors identifies REST

Control of chromosome stability by the beta-TrCP-REST-Mad2 axis

Notch signaling is required for normal prostatic epithelial cell proliferation and differentiation

Autophagy pathway is required for IL-6 induced neuroendocrine differentiation and chemoresistance of prostate cancer LNCaP cells

Bidirectional transport of amino acids regulates mTOR and autophagy

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26137-26151

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10.18632/ONCOTARGET.8433

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