IL-10 deficiency unleashes an influenza-specific Th17 response and enhances survival against high-dose challenge. - Wikidata - wikimedia - TriplyDB

IL-10 deficiency unleashes an influenza-specific Th17 response and enhances survival against high-dose challenge.

IL-10 deficiency unleashes an influenza-specific Th17 response and enhances survival against high-dose challenge.

http://www.wikidata.org/entity/Q37297787

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Evaluation of the sublingual route for administration of influenza H5N1 virosomes in combination with the bacterial second messenger c-di-GMP A novel inactivated intranasal respiratory syncytial virus vaccine promotes viral clearance without Th2 associated vaccine-enhanced disease New Insights into the Generation of CD4 Memory May Shape Future Vaccine Strategies for Influenza Bacterial-viral load and the immune response in stable and exacerbated COPD: significance and therapeutic prospects Influenza A Virus Infection, Innate Immunity, and Childhood Herpesvirus exploitation of host immune inhibitory pathways Regulating the adaptive immune response to respiratory virus infection Randomized controlled ferret study to assess the direct impact of 2008-09 trivalent inactivated influenza vaccine on A(H1N1)pdm09 disease risk Pulmonary infection with influenza A virus induces site-specific germinal center and T follicular helper cell responses Allergic airway disease in mice alters T and B cell responses during an acute respiratory poxvirus infection Type I IFN signaling facilitates the development of IL-10-producing effector CD8+ T cells during murine influenza virus infection.IL-21R signaling suppresses IL-17+ gamma delta T cell responses and production of IL-17 related cytokines in the lung at steady state and after Influenza A virus infection.Influenza virus-induced lung inflammation was modulated by cigarette smoke exposure in mice.The effector T cell response to influenza infection Primary and long-term B-cell responses in the upper airway and lung after influenza A virus infection.Novel pandemic influenza A (H1N1) and community-associated methicillin-resistant Staphylococcus aureus pneumonia.Why is coinfection with influenza virus and bacteria so difficult to control?Pregnancy outcome and clinical status of gilts following experimental infection by H1N2, H3N2 and H1N1pdm09 influenza A viruses during the last month of gestation.Th1 and Th17 hypercytokinemia as early host response signature in severe pandemic influenza.Host response to influenza virus: protection versus immunopathology.Postinfluenza bacterial pneumonia: host defenses gone awry.Host adaptive immunity deficiency in severe pandemic influenza.Role of host immune response and viral load in the differential outcome of pandemic H1N1 (2009) influenza virus infection in Indian patients Hallmarks of CD4 T cell immunity against influenza T cell responses during influenza infection: getting and keeping control.Differential host determinants contribute to the pathogenesis of 2009 pandemic H1N1 and human H5N1 influenza A viruses in experimental mouse models.The mucosal and systemic immune responses elicited by a chitosan-adjuvanted intranasal influenza H5N1 vaccine.Synergistic stimulation of type I interferons during influenza virus coinfection promotes Streptococcus pneumoniae colonization in mice.Changes in cytokine levels and NK cell activation associated with influenza.Mannose-binding lectin contributes to deleterious inflammatory response in pandemic H1N1 and avian H9N2 infection.Intensive cytokine induction in pandemic H1N1 influenza virus infection accompanied by robust production of IL-10 and IL-6 Role of T cell immunity in recovery from influenza virus infection High levels of virus-specific CD4+ T cells predict severe pandemic influenza A virus infection.Memory CD4 T cell-mediated immunity against influenza A virus: more than a little helpful.CD28 and ITK signals regulate autoreactive T cell trafficking.CD4+ T cell subset differentiation and avidity setpoint are dictated by the interplay of cytokine and antigen mediated signals A critical role of IL-17 in modulating the B-cell response during H5N1 influenza virus infection Timed action of IL-27 protects from immunopathology while preserving defense in influenza α4-integrins control viral meningoencephalitis through differential recruitment of T helper cell subsets.Prophylactic administration of bacterially derived immunomodulators improves the outcome of influenza virus infection in a murine model.

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IL-10 deficiency unleashes an influenza-specific Th17 response and enhances survival against high-dose challenge.

http://www.wikidata.org/entity/Q37297787

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scientific article published on June 2009

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JIMMUNOL.0900657

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Journal of Immunology

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Amanda Buck

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Gail Huston

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Hiromasa Hamada

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John P Dibble

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Jonathan D Curtis

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K Kai McKinstry

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Michael Tighe

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Richard W Dutton

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Stewart Sell

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Tara M Strutt

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P2860

Interleukin-10 and the interleukin-10 receptor

TGF-beta-induced Foxp3 inhibits T(H)17 cell differentiation by antagonizing RORgammat function

Interleukin-1 is responsible for acute lung immunopathology but increases survival of respiratory influenza virus infection

The biological functions of T helper 17 cell effector cytokines in inflammation

Tumor-specific Th17-polarized cells eradicate large established melanoma

Specific microbiota direct the differentiation of IL-17-producing T-helper cells in the mucosa of the small intestine

Interleukin-10 determines viral clearance or persistence in vivo

Determination of lymphocyte division by flow cytometry

Fatal outcome of human influenza A (H5N1) is associated with high viral load and hypercytokinemia

Interleukin-10-deficient mice develop chronic enterocolitis

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10.4049/JIMMUNOL.0900657

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