Amyloid beta-induced glycogen synthase kinase 3β phosphorylated VDAC1 in Alzheimer's disease: implications for synaptic dysfunction and neuronal damage
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Alterations in Mitochondrial Quality Control in Alzheimer's DiseaseParkin-mediated mitophagy in mutant hAPP neurons and Alzheimer's disease patient brainsMitochondrial VDAC1: A Key Gatekeeper as Potential Therapeutic TargetThe Voltage-dependent Anion Channel 1 Mediates Amyloid β Toxicity and Represents a Potential Target for Alzheimer Disease Therapy.BRCA1 and p53 tumor suppressor molecules in Alzheimer's disease.Common Polymorphisms in the GSK3β Gene May Contribute to the Pathogenesis of Alzheimer Disease: A Meta-Analysis.The Ca(2+) channel inhibitor 2-APB reverses β-amyloid-induced LTP deficit in hippocampus by blocking BAX and caspase-3 hyperactivationHow the Wnt signaling pathway protects from neurodegeneration: the mitochondrial scenarioWnt Signaling Prevents the Aβ Oligomer-Induced Mitochondrial Permeability Transition Pore Opening Preserving Mitochondrial Structure in Hippocampal Neurons.Multiple faces of dynamin-related protein 1 and its role in Alzheimer's disease pathogenesisS-Nitrosylation in Alzheimer's disease.Cell-based Models To Investigate Tau Aggregation.A Mitocentric View of Alzheimer's Disease.Amyloid beta modulators and neuroprotection in Alzheimer's disease: a critical appraisal.Dynamics of diabetes and obesity: Epidemiological perspective.Voltage-Dependent Anion Channel 1 As an Emerging Drug Target for Novel Anti-Cancer Therapeutics.Plasmalemmal VDAC-1 corroborated as amyloid Aß-receptor.Antioxidative effects of ethyl 2-(3-(benzo[d]thiazol-2-yl)ureido)acetate against amyloid β-induced oxidative cell death via NF-κB, GSK-3β and β-catenin signaling pathways in cultured cortical neurons.2-Cyclopropylimino-3-methyl-1,3-thiazoline hydrochloride protects against beta-amyloid-induced activation of the apoptotic cascade in cultured cortical neurons.Novel function of ceramide for regulation of mitochondrial ATP release in astrocytes.Guanosine monophosphate reductase 1 is a potential therapeutic target for Alzheimer's disease.Reduction in Aβ-induced cell death in the hippocampus of 17β-estradiol-treated female rats is associated with an increase in IGF-I signaling and somatostatinergic tone.AMPK is activated early in cerebellar granule cells undergoing apoptosis and influences VADC1 phosphorylation status and activity.GSK3β Regulates Brain Energy Metabolism.Alzheimer's Disease and Type 2 Diabetes: A Critical Assessment of the Shared Pathological Traits.Early Life Stress and Epigenetics in Late-onset Alzheimer's Dementia: A Systematic ReviewThe Akt pathway in oncology therapy and beyond (Review)
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Amyloid beta-induced glycogen synthase kinase 3β phosphorylated VDAC1 in Alzheimer's disease: implications for synaptic dysfunction and neuronal damage
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 June 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Amyloid beta-induced glycogen ...... ysfunction and neuronal damage
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Amyloid beta-induced glycogen ...... sfunction and neuronal damage.
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type
label
Amyloid beta-induced glycogen ...... ysfunction and neuronal damage
@en
Amyloid beta-induced glycogen ...... sfunction and neuronal damage.
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prefLabel
Amyloid beta-induced glycogen ...... ysfunction and neuronal damage
@en
Amyloid beta-induced glycogen ...... sfunction and neuronal damage.
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P2860
P1476
Amyloid beta-induced glycogen ...... ysfunction and neuronal damage
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P2093
P Hemachandra Reddy
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P304
P356
10.1016/J.BBADIS.2013.06.012
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P577
2013-06-28T00:00:00Z