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Leukocyte lipid body formation and eicosanoid generation: cyclooxygenase-independent inhibition by aspirin

Leukocyte lipid body formation and eicosanoid generation: cyclooxygenase-independent inhibition by aspirin

http://www.wikidata.org/entity/Q37310743

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Activation of human eosinophils through leukocyte immunoglobulin-like receptor 7 The antithrombotic profile of aspirin. Aspirin resistance, or simply failure?Diethylcarbamazine activity against Brugia malayi microfilariae is dependent on inducible nitric-oxide synthase and the cyclooxygenase pathway Host Lipid Bodies as Platforms for Intracellular Survival of Protozoan Parasites Lipid Body Organelles within the Parasite Trypanosoma cruzi: A Role for Intracellular Arachidonic Acid Metabolism The internal architecture of leukocyte lipid body organelles captured by three-dimensional electron microscopy tomography Biology and pathobiology of lipid droplets and their potential role in the protection of the organ of Corti.Single-cell Raman and fluorescence microscopy reveal the association of lipid bodies with phagosomes in leukocytes.Induction of endothelial cell cytoplasmic lipid bodies during hypoxia.Novel translocation responses of cytosolic phospholipase A2alpha fluorescent proteins.B-1 cells modulate the murine macrophage response to Leishmania major infection.A new fluorescence-based method identifies protein phosphatases regulating lipid droplet metabolism.Fatty acids from very low-density lipoprotein lipolysis products induce lipid droplet accumulation in human monocytes.Lipid bodies in inflammatory cells: structure, function, and current imaging techniques.Oleic acid inhibits lung Na/K-ATPase in mice and induces injury with lipid body formation in leukocytes and eicosanoid production.Lipid body accumulation alters calcium signaling dynamics in immune cells.Eosinophils as a novel cell source of prostaglandin D2: autocrine role in allergic inflammation.Co-compartmentalization of MAP kinases and cytosolic phospholipase A2 at cytoplasmic arachidonate-rich lipid bodies.Reduced plasma nonesterified fatty acid levels and the advent of an acute lung injury in mice after intravenous or enteral oleic acid administration Adipose triglyceride lipase acts on neutrophil lipid droplets to regulate substrate availability for lipid mediator synthesis.Mechanisms of leukocyte lipid body formation and function in inflammation.Hyperosmolarity-induced lipid droplet formation depends on ceramide production by neutral sphingomyelinase 2.Eosinophil lipid bodies: specific, inducible intracellular sites for enhanced eicosanoid formation.Oleic acid induces lung injury in mice through activation of the ERK pathway.In Vitro and In Vivo Trypanosomicidal Action of Novel Arylimidamides against Trypanosoma cruzi.Allergic challenge-elicited lipid bodies compartmentalize in vivo leukotriene C4 synthesis within eosinophils.Roles and origins of leukocyte lipid bodies: proteomic and ultrastructural studies.Multi-Target Approaches in Colon Cancer Chemoprevention Based on Systems Biology of Tumor Cell-Signaling.Leukocyte lipid bodies - Biogenesis and functions in inflammation RNAi-based strategies for cyclooxygenase-2 inhibition in cancer.Inhibition of neuroblastoma cell proliferation with omega-3 fatty acids and treatment of a murine model of human neuroblastoma using a diet enriched with omega-3 fatty acids in combination with sunitinib.LEUKOCYTE LIPID BODIES - STRUCTURE AND FUNCTION AS "EICOSASOMES".CD14-dependent internalization and metabolism of extracellular phosphatidylinositol by monocytes.Impairment of neutrophil migration to remote inflammatory site during lung histoplasmosis.Cyclo-oxygenase and colon cancer: clues to the aspirin effect?Activation of J77A.1 macrophages by three phospholipases A2 isolated from Bothrops atrox snake venom.Group IVA phospholipase A2 is necessary for the biogenesis of lipid droplets.Intestinal inflammation requires FOXO3 and prostaglandin E2-dependent lipogenesis and elevated lipid droplets.Host cell lipid bodies triggered by Trypanosoma cruzi infection and enhanced by the uptake of apoptotic cells are associated with prostaglandin E₂ generation and increased parasite growth.Mechanisms of formation and function of eosinophil lipid bodies: inducible intracellular sites involved in arachidonic acid metabolism.

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Leukocyte lipid body formation and eicosanoid generation: cyclooxygenase-independent inhibition by aspirin

http://www.wikidata.org/entity/Q37310743

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on October 1996

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Leukocyte lipid body formation ...... ependent inhibition by aspirin

@en

Leukocyte lipid body formation ...... pendent inhibition by aspirin.

@nl

type

label

Leukocyte lipid body formation ...... ependent inhibition by aspirin

@en

Leukocyte lipid body formation ...... pendent inhibition by aspirin.

@nl

prefLabel

Leukocyte lipid body formation ...... ependent inhibition by aspirin

@en

Leukocyte lipid body formation ...... pendent inhibition by aspirin.

@nl

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PNAS.93.20.11091

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Proceedings of the National Academy of Sciences of the United States of America

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Leukocyte lipid body formation ...... ependent inhibition by aspirin

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P2093

J L Payne

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P F Weller

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P T Bozza

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R Langenbach

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S G Morham

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P2860

Mechanisms of platelet-activating factor-induced lipid body formation: requisite roles for 5-lipoxygenase and de novo protein synthesis in the compartmentalization of neutrophil lipids

Inhibition of prostaglandin synthesis as a mechanism of action for aspirin-like drugs

Cytoplasmic lipid bodies of human eosinophils. Subcellular isolation and analysis of arachidonate incorporation.

Mechanism of selective inhibition of the inducible isoform of prostaglandin G/H synthase.

Glycogen accumulation in polymorphonuclear leukocytes, and other intracellular alterations that occur during inflammation.

Migration of human inflammatory cells into the lung results in the remodeling of arachidonic acid into a triglyceride pool.

Cytoplasmic lipid bodies of neutrophils: formation induced by cis-unsaturated fatty acids and mediated by protein kinase C.

Selective cumulative inhibition of platelet thromboxane production by low-dose aspirin in healthy subjects.

Modes of action of aspirin-like drugs.

Pharmacology of prostaglandin endoperoxide synthase isozymes-1 and -2.

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11091-11096

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10.1073/PNAS.93.20.11091

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93

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Oliver Smithies

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1996-10-01T00:00:00Z

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14350753

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8855314

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38289

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