HSV gene transfer in the treatment of chronic pain. - Wikidata - wikimedia - TriplyDB

HSV gene transfer in the treatment of chronic pain.

HSV gene transfer in the treatment of chronic pain.

http://www.wikidata.org/entity/Q37310900

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[EXPRESS] Intrathecal administration of human bone marrow mesenchymal stem cells genetically modified with human proenkephalin gene decrease nociceptive pain in neuropathic rats.Development of viral vectors for gene therapy for chronic pain

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HSV gene transfer in the treatment of chronic pain.

http://www.wikidata.org/entity/Q37310900

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on October 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

HSV gene transfer in the treatment of chronic pain.

@en

HSV gene transfer in the treatment of chronic pain.

@nl

type

label

HSV gene transfer in the treatment of chronic pain.

@en

HSV gene transfer in the treatment of chronic pain.

@nl

prefLabel

HSV gene transfer in the treatment of chronic pain.

@en

HSV gene transfer in the treatment of chronic pain.

@nl

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Acta Physiologica Sinica

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HSV gene transfer in the treatment of chronic pain

@en

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David J Fink

http://www.w3.org/2001/XMLSchema#string

Marina Mata

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P2860

HSV-mediated expression of interleukin-4 in dorsal root ganglion neurons reduces neuropathic pain.

Molecular mechanisms of nociception

Utilizing brain imaging for analgesic drug development.

Gene transfer of glutamic acid decarboxylase reduces neuropathic pain.

Neuronal plasticity and signal transduction in nociceptive neurons: implications for the initiation and maintenance of pathological pain.

Herpes vector-mediated gene transfer in treatment of diseases of the nervous system.

Antinociceptive effect of a genomic herpes simplex virus-based vector expressing human proenkephalin in rat dorsal root ganglion.

Transgene-mediated enkephalin release enhances the effect of morphine and evades tolerance to produce a sustained antiallodynic effect in neuropathic pain.

Peripherally delivered glutamic acid decarboxylase gene therapy for spinal cord injury pain.

Release of GABA from sensory neurons transduced with a GAD67-expressing vector occurs by non-vesicular mechanisms.

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610-616

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scholarly article

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5

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60

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2008-10-01T00:00:00Z

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18958369

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2586061

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