miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.
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MicroRNAs as regulators of metabolic disease: pathophysiologic significance and emerging role as biomarkers and therapeuticsThe role of miR-200a in mammalian epithelial cell transformation.Mechanisms Linking Inflammation to Insulin ResistanceMiR-19a regulates PTEN expression to mediate glycogen synthesis in hepatocytes.Protein phosphatase 4 (PP4) functions as a critical regulator in tumor necrosis factor (TNF)-α-induced hepatic insulin resistance.MicroRNA-20a-5p contributes to hepatic glycogen synthesis through targeting p63 to regulate p53 and PTEN expression.Reduced miR-200b and miR-200c expression contributes to abnormal hepatic lipid accumulation by stimulating JUN expression and activating the transcription of srebp1.Hepatic MiR-291b-3p Mediated Glucose Metabolism by Directly Targeting p65 to Upregulate PTEN Expression.Adipocyte miR-200b/a/429 ablation in mice leads to high-fat-diet-induced obesity.Suppressors of cytokine signaling (SOCS) and type 2 diabetes.Crosstalk between skin inflammation and adipose tissue-derived products: pathogenic evidence linking psoriasis to increased adiposity.Role of miRNAs in the pathogenesis and susceptibility of diabetes mellitus.MicroRNA 152 regulates hepatic glycogenesis by targeting PTEN.Loss of miR-141/200c ameliorates hepatic steatosis and inflammation by reprogramming multiple signaling pathways in NASH.The Pathogenesis of Obesity-Associated Adipose Tissue Inflammation.Comparison of Methods for Analyzing Human Adipose Tissue Macrophage Content.Maternal chromium restriction induces insulin resistance in adult mice offspring through miRNA.
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miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 24 June 2013
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.
@en
miR-200s contribute to interleukin-6
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type
label
miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.
@en
miR-200s contribute to interleukin-6
@nl
prefLabel
miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.
@en
miR-200s contribute to interleukin-6
@nl
P2093
P2860
P356
P1476
miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.
@en
P2093
Hangxiang Zhang
Lilin Wang
Xiuqing Huang
P2860
P304
22596-22606
P356
10.1074/JBC.M112.423145
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P577
2013-06-24T00:00:00Z