miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes. - Wikidata - wikimedia - TriplyDB

miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.

miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.

http://www.wikidata.org/entity/Q37311926

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MicroRNAs as regulators of metabolic disease: pathophysiologic significance and emerging role as biomarkers and therapeutics The role of miR-200a in mammalian epithelial cell transformation.Mechanisms Linking Inflammation to Insulin Resistance MiR-19a regulates PTEN expression to mediate glycogen synthesis in hepatocytes.Protein phosphatase 4 (PP4) functions as a critical regulator in tumor necrosis factor (TNF)-α-induced hepatic insulin resistance.MicroRNA-20a-5p contributes to hepatic glycogen synthesis through targeting p63 to regulate p53 and PTEN expression.Reduced miR-200b and miR-200c expression contributes to abnormal hepatic lipid accumulation by stimulating JUN expression and activating the transcription of srebp1.Hepatic MiR-291b-3p Mediated Glucose Metabolism by Directly Targeting p65 to Upregulate PTEN Expression.Adipocyte miR-200b/a/429 ablation in mice leads to high-fat-diet-induced obesity.Suppressors of cytokine signaling (SOCS) and type 2 diabetes.Crosstalk between skin inflammation and adipose tissue-derived products: pathogenic evidence linking psoriasis to increased adiposity.Role of miRNAs in the pathogenesis and susceptibility of diabetes mellitus.MicroRNA 152 regulates hepatic glycogenesis by targeting PTEN.Loss of miR-141/200c ameliorates hepatic steatosis and inflammation by reprogramming multiple signaling pathways in NASH.The Pathogenesis of Obesity-Associated Adipose Tissue Inflammation.Comparison of Methods for Analyzing Human Adipose Tissue Macrophage Content.Maternal chromium restriction induces insulin resistance in adult mice offspring through miRNA.

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miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.

http://www.wikidata.org/entity/Q37311926

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 24 June 2013

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.

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miR-200s contribute to interleukin-6

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type

label

miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.

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miR-200s contribute to interleukin-6

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prefLabel

miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.

@en

miR-200s contribute to interleukin-6

@nl

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JBC.M112.423145

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Journal of Biological Chemistry

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miR-200s contribute to interleukin-6 (IL-6)-induced insulin resistance in hepatocytes.

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Dan Gao

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Hangxiang Zhang

http://www.w3.org/2001/XMLSchema#string

Jian Li

http://www.w3.org/2001/XMLSchema#string

Juan Jiao

http://www.w3.org/2001/XMLSchema#string

Lilin Wang

http://www.w3.org/2001/XMLSchema#string

Lin Dou

http://www.w3.org/2001/XMLSchema#string

Shu Wang

http://www.w3.org/2001/XMLSchema#string

Tao Shen

http://www.w3.org/2001/XMLSchema#string

Ting Zhao

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Xiuqing Huang

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P2860

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Control of glucose homeostasis and insulin sensitivity by the Let-7 family of microRNAs

The Lin28/let-7 axis regulates glucose metabolism

A mammalian microRNA expression atlas based on small RNA library sequencing

miR-375 maintains normal pancreatic alpha- and beta-cell mass

MicroRNAs: genomics, biogenesis, mechanism, and function

Conserved MicroRNA miR-8/miR-200 and its target USH/FOG2 control growth by regulating PI3K

Obesity-induced overexpression of miRNA-143 inhibits insulin-stimulated AKT activation and impairs glucose metabolism

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scholarly article

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10.1074/JBC.M112.423145

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2013-06-24T00:00:00Z

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