B cell-derived transforming growth factor-β1 expression limits the induction phase of autoimmune neuroinflammation. - Wikidata - wikimedia - TriplyDB

B cell-derived transforming growth factor-β1 expression limits the induction phase of autoimmune neuroinflammation.

B cell-derived transforming growth factor-β1 expression limits the induction phase of autoimmune neuroinflammation.

http://www.wikidata.org/entity/Q37314912

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Activation of human B cells negatively regulates TGF-β1 production.B Cell-Directed Therapeutics in Multiple Sclerosis: Rationale and Clinical Evidence.Deciphering the Role of B Cells in Multiple Sclerosis-Towards Specific Targeting of Pathogenic Function.Regulatory B cells: the cutting edge of immune tolerance in kidney transplantation.Murine splenic B cells express corticotropin-releasing hormone receptor 2 that affect their viability during a stress response.Automatic microscopic detection of mycobacteria in sputum: a proof-of-concept Regulatory B and T lymphocytes in multiple sclerosis: friends or foes?Reevaluation of Pluripotent Cytokine TGF-β3 in Immunity

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B cell-derived transforming growth factor-β1 expression limits the induction phase of autoimmune neuroinflammation.

http://www.wikidata.org/entity/Q37314912

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 October 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

B cell-derived transforming gr ...... autoimmune neuroinflammation.

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B cell-derived transforming gr ...... autoimmune neuroinflammation.

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type

label

B cell-derived transforming gr ...... autoimmune neuroinflammation.

@en

B cell-derived transforming gr ...... autoimmune neuroinflammation.

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prefLabel

B cell-derived transforming gr ...... autoimmune neuroinflammation.

@en

B cell-derived transforming gr ...... autoimmune neuroinflammation.

@nl

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Scientific Reports

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B cell-derived transforming gr ...... f autoimmune neuroinflammation

@en

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Kristbjörg Bjarnadóttir

http://www.w3.org/2001/XMLSchema#string

Mahdia Benkhoucha

http://www.w3.org/2001/XMLSchema#string

Martin S Weber

http://www.w3.org/2001/XMLSchema#string

Natalie L Payne

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Nicolas Molnarfi

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Patrice H Lalive

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P2860

The crystal structure of myelin oligodendrocyte glycoprotein, a key autoantigen in multiple sclerosis

B-cell depletion with rituximab in relapsing-remitting multiple sclerosis

TGF-beta signaling in dendritic cells is a prerequisite for the control of autoimmune encephalomyelitis.

The inhibitory effects of transforming growth factor-beta-1 (TGF-beta1) in autoimmune diseases.

Lipopolysaccharide-activated B cells down-regulate Th1 immunity and prevent autoimmune diabetes in nonobese diabetic mice.

Characterization of a rare IL-10-competent B-cell subset in humans that parallels mouse regulatory B10 cells.

B cells regulate autoimmunity by provision of IL-10.

Ocrelizumab in relapsing-remitting multiple sclerosis: a phase 2, randomised, placebo-controlled, multicentre trial.

Immunoglobulin class-switched B cells form an active immune axis between CNS and periphery in multiple sclerosis.

Experimental autoimmune encephalomyelitis induction in genetically B cell-deficient mice.

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34594

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scholarly article

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10.1038/SREP34594

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6

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Claude C A Bernard

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2016-10-06T00:00:00Z

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27708418

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