Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ. - Wikidata - wikimedia - TriplyDB

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

http://www.wikidata.org/entity/Q37319591

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Peroxisome Proliferator-Activated Receptors in Female Reproduction and Fertility Regulation of brown adipose tissue recruitment, metabolism and thermogenic function by peroxisome proliferator-activated receptor γ Obesity genetics in mouse and human: back and forth, and back again The brain and brown fat Commonalities in the Association between PPARG and Vitamin D Related with Obesity and Carcinogenesis The Complex Roles of Mechanistic Target of Rapamycin in Adipocytes and Beyond.PPARs in obesity-induced T2DM, dyslipidaemia and NAFLD.Selective Tissue Distribution Mediates Tissue-Dependent PPARγ Activation and Insulin Sensitization by INT131, a Selective PPARγ Modulator.Adipose tissue NAD+ biology in obesity and insulin resistance: From mechanism to therapy Regulation of insulin resistance and adiponectin signaling in adipose tissue by liver X receptor activation highlights a cross-talk with PPARγ.PPARγ and the global map of adipogenesis and beyond.Negative regulators of brown adipose tissue (BAT)-mediated thermogenesis Role of forkhead box protein A3 in age-associated metabolic decline.Thiazolidinediones and the promise of insulin sensitization in type 2 diabetes.Loss of Gsα early in the osteoblast lineage favors adipogenic differentiation of mesenchymal progenitors and committed osteoblast precursors.Fetal development of subcutaneous white adipose tissue is dependent on Zfp423.NPY signalling in early osteoblasts controls glucose homeostasis.TonEBP suppresses adipogenesis and insulin sensitivity by blocking epigenetic transition of PPARγ2.Can Brown Fat Win the Battle Against White Fat?The role of hepatic lipids in hepatic insulin resistance and type 2 diabetes Genetic Variation Determines PPARγ Function and Anti-diabetic Drug Response In Vivo.Distinct regulatory mechanisms governing embryonic versus adult adipocyte maturation.In utero growth restriction and catch-up adipogenesis after developmental di (2-ethylhexyl) phthalate exposure cause glucose intolerance in adult male rats following a high-fat dietary challenge.Impact of tamoxifen on adipocyte lineage tracing: Inducer of adipogenesis and prolonged nuclear translocation of Cre recombinase The glucocorticoid receptor: cause of or cure for obesity?Adipose tissue mTORC2 regulates ChREBP-driven de novo lipogenesis and hepatic glucose metabolism Raptor/mTORC1 loss in adipocytes causes progressive lipodystrophy and fatty liver disease.Lack of AKT in adipocytes causes severe lipodystrophy.The transcriptional coregulator GRIP1 controls macrophage polarization and metabolic homeostasis SRF and MKL1 Independently Inhibit Brown Adipogenesis.Nuclear receptors and metabolism: from feast to famine.Congenital lipodystrophies and dyslipidemias.PPARs and ERRs: molecular mediators of mitochondrial metabolism.Obesity-induced endoplasmic reticulum stress suppresses nuclear factor-Y expression.The multifaceted factor peroxisome proliferator-activated receptor γ (PPARγ) in metabolism, immunity, and cancer.Obesity-associated sympathetic overactivity in children and adolescents: the role of catecholamine resistance in lipid metabolism.The Role of Peroxisome Proliferator-Activated Receptor Gamma (PPARG) in Adipogenesis: Applying Knowledge from the Fish Aquaculture Industry to Biomedical Research.Pancreatic Islet Responses to Metabolic Trauma.Black rice (Oryza sativa L.) extracts induce osteoblast differentiation and protect against bone loss in ovariectomized rats.A dynamic CTCF chromatin binding landscape promotes DNA hydroxymethylation and transcriptional induction of adipocyte differentiation

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P2860

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

http://www.wikidata.org/entity/Q37319591

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 28 October 2013

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

@en

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

@nl

type

label

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

@en

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

@nl

prefLabel

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

@en

Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

@nl

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PNAS.1314863110

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Proceedings of the National Academy of Sciences of the United States of America

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Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ.

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Fenfen Wang

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Joanna R DiSpirito

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Lindsey C Peed

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Mitchell A Lazar

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Shannon E Mullican

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P2860

Dominant negative mutations in human PPARgamma associated with severe insulin resistance, diabetes mellitus and hypertension

Deletion of PPARgamma in adipose tissues of mice protects against high fat diet-induced obesity and insulin resistance

PPAR gamma is required for the differentiation of adipose tissue in vivo and in vitro

Leptin inhibits bone formation through a hypothalamic relay: a central control of bone mass

Stimulation of adipogenesis in fibroblasts by PPAR gamma 2, a lipid-activated transcription factor

An antidiabetic thiazolidinedione is a high affinity ligand for peroxisome proliferator-activated receptor gamma (PPAR gamma)

mPPAR gamma 2: tissue-specific regulator of an adipocyte enhancer

Adipocytes as regulators of energy balance and glucose homeostasis

PPAR gamma is required for placental, cardiac, and adipose tissue development

Adipokines in inflammation and metabolic disease

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10.1073/PNAS.1314863110

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2013-10-28T00:00:00Z

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