Polo-like kinases mediate cell survival in mitochondrial dysfunction. - Wikidata - wikimedia - TriplyDB

Polo-like kinases mediate cell survival in mitochondrial dysfunction.

Polo-like kinases mediate cell survival in mitochondrial dysfunction.

http://www.wikidata.org/entity/Q37321107

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Mitochondrial disulfide relay mediates translocation of p53 and partitions its subcellular activity The Functional Significance of Posttranslational Modifications on Polo-Like Kinase 1 Revealed by Chemical Genetic Complementation Selective and brain-permeable polo-like kinase-2 (Plk-2) inhibitors that reduce α-synuclein phosphorylation in rat brain Structural basis for the inhibition of Polo-like kinase 1 A translatable predictor of human radiation exposure Dasatinib synergises with irinotecan to suppress hepatocellular carcinoma via inhibiting the protein synthesis of PLK1.PLK2 Plays an Essential Role in High D-Glucose-Induced Apoptosis, ROS Generation and Inflammation in Podocytes.Polo-like kinase 2 activates an antioxidant pathway to promote the survival of cells with mitochondrial dysfunction.Genetic determinants at the interface of cancer and neurodegenerative disease.Dynamics of the transcriptome response of cultured human embryonic stem cells to ionizing radiation exposure.p53, aerobic metabolism, and cancer.Mitochondrial respiration protects against oxygen-associated DNA damage.Polo-like kinase 2 acting as a promoter in human tumor cells with an abundance of TAp73.Bafilomycin A1 activates HIF-dependent signalling in human colon cancer cells via mitochondrial uncoupling PLK2 phosphorylates and inhibits enriched TAp73 in human osteosarcoma cells Nuclear pore protein NUP88 activates anaphase-promoting complex to promote aneuploidy.Standardized bioenergetic profiling of adult mouse cardiomyocytes.Differential transcriptomic analysis of spontaneous lung tumors in B6C3F1 mice: comparison to human non-small cell lung cancer.AAV-mediated gene targeting methods for human cells Inhibiting mitochondrial respiration prevents cancer in a mouse model of Li-Fraumeni syndrome.A novel form of necrosis, TRIAD, occurs in human Huntington's disease Implication of Alpha-Synuclein Phosphorylation at S129 in Synucleinopathies: What Have We Learned in the Last Decade?Serum inducible kinase is a positive regulator of cortical dendrite development and is required for BDNF-promoted dendritic arborization.Measurement of cell respiration and oxygenation in standard multichannel biochips using phosphorescent O2-sensitive probes.E46K α-synuclein pathological mutation causes cell-autonomous toxicity without altering protein turnover or aggregation.

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Polo-like kinases mediate cell survival in mitochondrial dysfunction.

http://www.wikidata.org/entity/Q37321107

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 11 August 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Polo-like kinases mediate cell survival in mitochondrial dysfunction.

@en

Polo-like kinases mediate cell survival in mitochondrial dysfunction.

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type

label

Polo-like kinases mediate cell survival in mitochondrial dysfunction.

@en

Polo-like kinases mediate cell survival in mitochondrial dysfunction.

@nl

prefLabel

Polo-like kinases mediate cell survival in mitochondrial dysfunction.

@en

Polo-like kinases mediate cell survival in mitochondrial dysfunction.

@nl

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PNAS.0904229106

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Proceedings of the National Academy of Sciences of the United States of America

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Polo-like kinases mediate cell survival in mitochondrial dysfunction.

@en

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Ho Joong Sung

http://www.w3.org/2001/XMLSchema#string

Paul M Hwang

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Ping-Yuan Wang

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Satoaki Matoba

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Takumi Matsumoto

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Wenzhe Ma

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P2860

Fatal infantile cardioencephalomyopathy with COX deficiency and mutations in SCO2, a COX assembly gene

Polo-like kinase-1 is activated by aurora A to promote checkpoint recovery

Plk1 promotes nuclear translocation of human Cdc25C during prophase

Improved methods for the generation of human gene knockout and knockin cell lines

Facile methods for generating human somatic cell gene knockouts using recombinant adeno-associated viruses

A panel of isogenic human cancer cells suggests a therapeutic approach for cancers with inactivated p53

Silencing of the novel p53 target gene Snk/Plk2 leads to mitotic catastrophe in paclitaxel (taxol)-exposed cells

Role of Plk2 (Snk) in mouse development and cell proliferation

On the Origin of Cancer Cells

Bora and the kinase Aurora a cooperatively activate the kinase Plk1 and control mitotic entry

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14542-14546

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scholarly article

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10.1073/PNAS.0904229106

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