Magnitude and control of mitochondrial sensitivity to ADP.
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Parameter trajectory analysis to identify treatment effects of pharmacological interventionsComputational modeling of mitochondrial energy transduction.Adenine nucleotide translocase is acetylated in vivo in human muscle: Modeling predicts a decreased ADP affinity and altered control of oxidative phosphorylation.In-vivo31P-MRS of skeletal muscle and liver: A way for non-invasive assessment of their metabolism.Application of the principles of systems biology and Wiener's cybernetics for analysis of regulation of energy fluxes in muscle cells in vivo.Glucose sensing in the pancreatic beta cell: a computational systems analysis.Prediction of muscle energy states at low metabolic rates requires feedback control of mitochondrial respiratory chain activity by inorganic phosphate.A computational model of torque generation: neural, contractile, metabolic and musculoskeletal components.Similar mitochondrial activation kinetics in wild-type and creatine kinase-deficient fast-twitch muscle indicate significant Pi control of respiration.Impaired adaptability of in vivo mitochondrial energetics to acute oxidative insult in aged skeletal muscleOn the kinetics of anaerobic power.Short-term training alters the control of mitochondrial respiration rate before maximal oxidative ATP synthesis.The systems biology of mitochondrial fission and fusion and implications for disease and aging.Modelling in vivo creatine/phosphocreatine in vitro reveals divergent adaptations in human muscle mitochondrial respiratory control by ADP after acute and chronic exercise.Unchanged muscle fiber conduction velocity relates to mild acidosis during exhaustive bicycling.Comparison of in vivo postexercise phosphocreatine recovery and resting ATP synthesis flux for the assessment of skeletal muscle mitochondrial function.Slowed muscle oxygen uptake kinetics with raised metabolism are not dependent on blood flow or recruitment dynamics.Effects of exercise-induced intracellular acidosis on the phosphocreatine recovery kinetics: a 31P MRS study in three muscle groups in humans.Reaction-diffusion constraints in living tissue: effectiveness factors in skeletal muscle design.On-off asymmetries in oxygen consumption kinetics of single Xenopus laevis skeletal muscle fibres suggest higher-order control.Kinetic control of oxygen consumption during contractions in self-perfused skeletal muscle
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P2860
Magnitude and control of mitochondrial sensitivity to ADP.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 21 July 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Magnitude and control of mitochondrial sensitivity to ADP.
@en
Magnitude and control of mitochondrial sensitivity to ADP.
@nl
type
label
Magnitude and control of mitochondrial sensitivity to ADP.
@en
Magnitude and control of mitochondrial sensitivity to ADP.
@nl
prefLabel
Magnitude and control of mitochondrial sensitivity to ADP.
@en
Magnitude and control of mitochondrial sensitivity to ADP.
@nl
P2093
P2860
P50
P1476
Magnitude and control of mitochondrial sensitivity to ADP
@en
P2093
Jeroen A L Jeneson
Joep P J Schmitz
Nicole M A van den Broek
Peter A J Hilbers
P2860
P304
P356
10.1152/AJPENDO.00370.2009
P577
2009-07-21T00:00:00Z