Activation of estrogen receptor transfected into a receptor-negative breast cancer cell line decreases the metastatic and invasive potential of the cells.
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Direct visualization of the human estrogen receptor alpha reveals a role for ligand in the nuclear distribution of the receptorReciprocal regulation of extracellular matrix proteins and ovarian steroid activity in the mammary gland.How does the estrogen receptor work?Drug development against metastasis-related genes and their pathways: A rationale for cancer therapyPathways to tamoxifen resistance.Estrogen receptor ESR1 controls cell migration by repressing chemokine receptor CXCR4 in the zebrafish posterior lateral line systemThe ligand-mediated nuclear mobility and interaction with estrogen-responsive elements of estrogen receptors are subtype specific.Loss of estrogen receptor 1 enhances cervical cancer invasion.Estrogen receptor beta functions through nongenomic mechanisms in lung cancer cells.ER-alpha-cDNA as part of a bicistronic transcript gives rise to high frequency, long term, receptor expressing cell clones.Effects of human mesenchymal stem cells on ER-positive human breast carcinoma cells mediated through ER-SDF-1/CXCR4 crosstalk.ShRNA-mediated gene silencing of MTA1 influenced on protein expression of ER alpha, MMP-9, CyclinD1 and invasiveness, proliferation in breast cancer cell lines MDA-MB-231 and MCF-7 in vitroCytokine receptor CXCR4 mediates estrogen-independent tumorigenesis, metastasis, and resistance to endocrine therapy in human breast cancer.Cellular reprogramming by the conjoint action of ERα, FOXA1, and GATA3 to a ligand-inducible growth state.Generation of stable reporter breast cancer cell lines for the identification of ER subtype selective ligands.Inhibition of skeletal metastasis by ectopic ERalpha expression in ERalpha-negative human breast cancer cell lines.Expression of prolactin and prolactin receptor in human breast carcinoma. Evidence for an autocrine/paracrine loopEffects of SDF-1-CXCR4 signaling on microRNA expression and tumorigenesis in estrogen receptor-alpha (ER-α)-positive breast cancer cells.IL-8 expression and its possible relationship with estrogen-receptor-negative status of breast cancer cells.ER beta inhibits proliferation and invasion of breast cancer cellsp38gamma mitogen-activated protein kinase integrates signaling crosstalk between Ras and estrogen receptor to increase breast cancer invasionEstrogen receptor-alpha overexpression suppresses 17beta-estradiol-mediated vascular endothelial growth factor expression and activation of survival kinasesRole of estrogens and their receptors in adhesion and invasiveness of breast cancer cells.TARGETING THE GENOTOXIC EFFECTS OF ESTROGENS.DNA hypermethylation profiles in squamous cell carcinoma of the vulva.Estrogen receptor alpha is cell cycle-regulated and regulates the cell cycle in a ligand-dependent fashionSignaling Transduction Network Mediated by Tumor Suppressor/Susceptibility Genes in NPC.The metastasis inducer CCN1 (CYR61) activates the fatty acid synthase (FASN)-driven lipogenic phenotype in breast cancer cells.Designer monotransregulators provide a basis for a transcriptional therapy for de novo endocrine-resistant breast cancer.Estrogen receptor alpha inhibits senescence-like phenotype and facilitates transformation induced by oncogenic ras in human mammary epithelial cells.Nucleophosmin/B23 is a negative regulator of estrogen receptor α expression via AP2γ in endometrial cancer cells.The histone deacetylase inhibitor trichostatin A alters microRNA expression profiles in apoptosis-resistant breast cancer cells.Lymphokine-activated killer cell susceptibility and adhesion molecule expression of multidrug resistant breast carcinoma.Estrogen-induced upregulation and 3'-UTR shortening of CDC6Promoter methylation in head and neck tumorigenesis.Estrogen receptors similarly mediate the effects of 17β-estradiol on cellular responses but differ in their potenciesInfluence of estradiol and triiodothyronine on breast cancer cell lines proliferation and expression of estrogen and thyroid hormone receptors.Genomic responses from the estrogen-responsive element-dependent signaling pathway mediated by estrogen receptor alpha are required to elicit cellular alterations.Gene expression profiling reveals that the regulation of estrogen-responsive element-independent genes by 17 beta-estradiol-estrogen receptor beta is uncoupled from the induction of phenotypic changes in cell models.Restoration of tamoxifen sensitivity in estrogen receptor-negative breast cancer cells: tamoxifen-bound reactivated ER recruits distinctive corepressor complexes.
P2860
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P2860
Activation of estrogen receptor transfected into a receptor-negative breast cancer cell line decreases the metastatic and invasive potential of the cells.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 1992
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Activation of estrogen recepto ...... vasive potential of the cells.
@en
Activation of estrogen recepto ...... vasive potential of the cells.
@nl
type
label
Activation of estrogen recepto ...... vasive potential of the cells.
@en
Activation of estrogen recepto ...... vasive potential of the cells.
@nl
prefLabel
Activation of estrogen recepto ...... vasive potential of the cells.
@en
Activation of estrogen recepto ...... vasive potential of the cells.
@nl
P2093
P2860
P356
P1476
Activation of estrogen recepto ...... vasive potential of the cells.
@en
P2093
P2860
P304
11538-11542
P356
10.1073/PNAS.89.23.11538
P407
P577
1992-12-01T00:00:00Z