Deficiency of thrombospondin-1 reduces Th17 differentiation and attenuates experimental autoimmune encephalomyelitis. - Wikidata - wikimedia - TriplyDB

Deficiency of thrombospondin-1 reduces Th17 differentiation and attenuates experimental autoimmune encephalomyelitis.

Deficiency of thrombospondin-1 reduces Th17 differentiation and attenuates experimental autoimmune encephalomyelitis.

http://www.wikidata.org/entity/Q37327995

about

Thrombospondin1 in tissue repair and fibrosis: TGF-β-dependent and independent mechanisms Shared epitope-antagonistic ligands: a new therapeutic strategy in mice with erosive arthritis.Ovariectomized OVA-sensitized mice display increased frequency of CD4(+)Foxp3(+) T regulatory cells in the periphery Dendritic cell-derived thrombospondin-1 is critical for the generation of the ocular surface Th17 response to desiccating stress.Angiotensin II sustains brain inflammation in mice via TGF-beta Blockade of TSP1-dependent TGF-β activity reduces renal injury and proteinuria in a murine model of diabetic nephropathy.Pearls in autoimmunity Thrombospondin-1 production is enhanced by Porphyromonas gingivalis lipopolysaccharide in THP-1 cells.Environmental conditioning in the control of macrophage thrombospondin-1 production.Identification and characterization of latency-associated peptide-expressing γδ T cells Discovery of novel disease-specific and membrane-associated candidate markers in a mouse model of multiple sclerosis.Role of thrombospondin 1 in liver diseases.Thrombospondin-1 as a Paradigm for the Development of Antiangiogenic Agents Endowed with Multiple Mechanisms of Action.Advantages of multiplex proteomics in clinical immunology: the case of rheumatoid arthritis: novel IgXPLEX™: planar microarray diagnosis.Thrombospondin-1 deficiency causes a shift from fibroproliferative to inflammatory kidney disease and delays onset of renal failure.Reproductive immunology: current status and future directions (part I).[Role of thrombospondin-1 in the development of kidney diseases].Exacerbated experimental autoimmune encephalomyelitis in mast-cell-deficient Kit W-sh/W-sh mice.Thrombospondin-1 regulation of latent TGF-β activation: A therapeutic target for fibrotic disease.Modulation of cellular immunity by antibodies against calreticulin.

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Deficiency of thrombospondin-1 reduces Th17 differentiation and attenuates experimental autoimmune encephalomyelitis.

http://www.wikidata.org/entity/Q37327995

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on February 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Deficiency of thrombospondin-1 ...... autoimmune encephalomyelitis.

@en

Deficiency of thrombospondin-1 ...... autoimmune encephalomyelitis.

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Deficiency of thrombospondin-1 ...... autoimmune encephalomyelitis.

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Deficiency of thrombospondin-1 ...... autoimmune encephalomyelitis.

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prefLabel

Deficiency of thrombospondin-1 ...... autoimmune encephalomyelitis.

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Deficiency of thrombospondin-1 ...... autoimmune encephalomyelitis.

@nl

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J.JAUT.2008.12.004

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Journal of Autoimmunity

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Deficiency of thrombospondin-1 ...... autoimmune encephalomyelitis.

@en

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Bing Zhu

http://www.w3.org/2001/XMLSchema#string

Jean Pierre Schatzmann Peron

http://www.w3.org/2001/XMLSchema#string

Jose L Vega

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Kaiyong Yang

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Luiz Vicente Rizzo

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Muhamed Hadzipasic

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Sharmila Masli

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Yijun Carrier

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P2860

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Receptor communication within the lymphocyte plasma membrane: a role for the thrombospondin family of matricellular proteins

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

T cell-produced transforming growth factor-beta1 controls T cell tolerance and regulates Th1- and Th17-cell differentiation

Thrombospondin-1 is a major activator of TGF-beta1 in vivo

Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis

Increased expression of interleukin 17 in inflammatory bowel disease

The functions of thrombospondin-1 and-2.

The cell biology of thrombospondin-1.

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94-103

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scholarly article

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10.1016/J.JAUT.2008.12.004

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2

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32

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Howard L. Weiner

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2009-02-01T00:00:00Z

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23963401

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19181483

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experimental autoimmune encephalomyelitis

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2735759

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