γδ T cells are essential effectors of type 1 diabetes in the nonobese diabetic mouse model. - Wikidata - wikimedia - TriplyDB

γδ T cells are essential effectors of type 1 diabetes in the nonobese diabetic mouse model.

γδ T cells are essential effectors of type 1 diabetes in the nonobese diabetic mouse model.

http://www.wikidata.org/entity/Q37331392

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MicroRNAs: Novel Players in the Dialogue between Pancreatic Islets and Immune System in Autoimmune Diabetes The streetlight effect in type 1 diabetes γδ T cells recognize the insulin B:9-23 peptide antigen when it is dimerized through thiol oxidation.Gut microbiota promote hematopoiesis to control bacterial infection Host microbiota constantly control maturation and function of microglia in the CNS.Potential role of IL-17-producing iNKT cells in type 1 diabetes.Communicating systems in the body: how microbiota and microglia cooperate.γδ T Cell-Mediated Immune Responses in Disease and Therapy γδT Cells and Related Diseases.IL-17+ γδ T cells as kick-starters of inflammation.Chemotaxis and Immunoregulatory Function of Cardiac γδ T Cells in Dystrophin-Deficient Mice.Loss of TCR responsiveness during thymic education imprints the 'innate' signature on γδ T cells.IL-23 drives differentiation of peripheral γδ17 T cells from adult bone marrow-derived precursors.HEB is required for the specification of fetal IL-17-producing γδ T cells.

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γδ T cells are essential effectors of type 1 diabetes in the nonobese diabetic mouse model.

http://www.wikidata.org/entity/Q37331392

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 26 April 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

γδ T cells are essential effec ...... nonobese diabetic mouse model.

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γδ T cells are essential effec ...... nonobese diabetic mouse model.

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type

label

γδ T cells are essential effec ...... nonobese diabetic mouse model.

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γδ T cells are essential effec ...... nonobese diabetic mouse model.

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prefLabel

γδ T cells are essential effec ...... nonobese diabetic mouse model.

@en

γδ T cells are essential effec ...... nonobese diabetic mouse model.

@nl

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JIMMUNOL.1203502

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Journal of Immunology

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γδ T cells are essential effec ...... nonobese diabetic mouse model

@en

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Andrea S L Wong

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Jayne S Danska

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Liping Geng

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Steve Mortin-Toth

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P2860

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

[gamma][delta] cells: a right time and a right place for a conserved third way of protection

Autoreactivity by design: innate B and T lymphocytes

Thymic selection determines gammadelta T cell effector fate: antigen-naive cells make interleukin-17 and antigen-experienced cells make interferon gamma

Thymic maturation determines gammadelta T cell function, but not their antigen specificities

Skint-1 identifies a common molecular mechanism for the development of interferon-γ-secreting versus interleukin-17-secreting γδ T cells

Innate IL-17-producing cells: the sentinels of the immune system.

Gamma delta T cell receptors confer autonomous responsiveness to the insulin-peptide B:9-23.

Interleukin-2 gene variation impairs regulatory T cell function and causes autoimmunity.

Two genetic loci regulate T cell-dependent islet inflammation and drive autoimmune diabetes pathogenesis

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5392-5401

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scholarly article

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10.4049/JIMMUNOL.1203502

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11

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190

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Adrian Clive Hayday

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2013-04-26T00:00:00Z

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236461135

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23626013

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type-1 diabetes

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3836168

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