Sialic acid catabolism confers a competitive advantage to pathogenic vibrio cholerae in the mouse intestine.
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Bacterial Communities: Interactions to ScaleInsights into Vibrio cholerae intestinal colonization from monitoring fluorescently labeled bacteriaStructural insights into the regulation of sialic acid catabolism by the Vibrio vulnificus transcriptional repressor NanRBacterial periplasmic sialic acid-binding proteins exhibit a conserved binding siteClostridium perfringens Sialidases: Potential Contributors to Intestinal Pathogenesis and Therapeutic TargetsVibrio cholerae in an Historically Cholera-Free CountryGenomic and phenotypic characterization of Vibrio cholerae non-O1 isolates from a US Gulf Coast cholera outbreak.Efficient responses to host and bacterial signals during Vibrio cholerae colonizationExcision dynamics of Vibrio pathogenicity island-2 from Vibrio cholerae: role of a recombination directionality factor VefAMetabolism of sialic acid by Bifidobacterium breve UCC2003.A strategy for neuraminidase inhibitors using mechanism-based labeling information.Catabolism of host-derived compounds during extracellular bacterial infections.RNA-Seq-based monitoring of infection-linked changes in Vibrio cholerae gene expression.The Entner-Doudoroff pathway is obligatory for gluconate utilization and contributes to the pathogenicity of Vibrio cholerae.Tripartite ATP-independent periplasmic (TRAP) transporters in bacteria and archaea.Milk sialyllactose influences colitis in mice through selective intestinal bacterial colonization.Intestinal Colonization Dynamics of Vibrio cholerae.Bacterial catabolism of nonulosonic (sialic) acid and fitness in the gut.Genotypic diversity and virulence characteristics of clinical and environmental Vibrio vulnificus isolates from the Baltic Sea region.An infant-associated bacterial commensal utilizes breast milk sialyloligosaccharides.Utilization of major fucosylated and sialylated human milk oligosaccharides by isolated human gut microbesCholera: Environmental Reservoirs and Impact on Disease Transmission.Are pathogenic bacteria just looking for food? Metabolism and microbial pathogenesis.Non-O1/non-O139 Vibrio cholerae carrying multiple virulence factors and V. cholerae O1 in the Chesapeake Bay, Maryland.Roles of the sodium-translocating NADH:quinone oxidoreductase (Na+-NQR) on vibrio cholerae metabolism, motility and osmotic stress resistance.Ecology and genetic structure of a northern temperate Vibrio cholerae population related to toxigenic isolates.Cooperative regulation of the Vibrio vulnificus nan gene cluster by NanR protein, cAMP receptor protein, and N-acetylmannosamine 6-phosphateSialylated Milk Oligosaccharides Promote Microbiota-Dependent Growth in Models of Infant Undernutrition.Sialic acid catabolism and transport gene clusters are lineage specific in Vibrio vulnificusGenomic diversity of 2010 Haitian cholera outbreak strains.Genomic and Evolutionary Analysis of Two Salmonella enterica Serovar Kentucky Sequence Types Isolated from Bovine and Poultry Sources in North America.Deacetylation of sialic acid by esterases potentiates pneumococcal neuraminidase activity for mucin utilization, colonization and virulence.Nutrient-deprived cancer cells preferentially use sialic acid to maintain cell surface glycosylation.Carbohydrate utilization by enterohaemorrhagic Escherichia coli O157:H7 in bovine intestinal contentCatabolism of N-acetylneuraminic acid, a fitness function of the food-borne lactic acid bacterium Lactobacillus sakei, involves two newly characterized proteins.Cloning, expression, purification, crystallization and preliminary X-ray diffraction studies of N-acetylneuraminate lyase from methicillin-resistant Staphylococcus aureus.Pathogenicity Island Cross Talk Mediated by Recombination Directionality Factors Facilitates Excision from the Chromosome.Sialic acid catabolism in Staphylococcus aureusDegradation, foraging, and depletion of mucus sialoglycans by the vagina-adapted Actinobacterium Gardnerella vaginalisCharacterization of undermethylated sites in Vibrio cholerae
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Sialic acid catabolism confers a competitive advantage to pathogenic vibrio cholerae in the mouse intestine.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 29 June 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Sialic acid catabolism confers ...... olerae in the mouse intestine.
@en
Sialic acid catabolism confers ...... olerae in the mouse intestine.
@nl
type
label
Sialic acid catabolism confers ...... olerae in the mouse intestine.
@en
Sialic acid catabolism confers ...... olerae in the mouse intestine.
@nl
prefLabel
Sialic acid catabolism confers ...... olerae in the mouse intestine.
@en
Sialic acid catabolism confers ...... olerae in the mouse intestine.
@nl
P2860
P356
P1476
Sialic acid catabolism confers ...... holerae in the mouse intestine
@en
P2093
E Fidelma Boyd
P2860
P304
P356
10.1128/IAI.00279-09
P407
P577
2009-06-29T00:00:00Z