Complement C3 plays an essential role in the control of opportunistic fungal infections. - Wikidata - wikimedia - TriplyDB

Complement C3 plays an essential role in the control of opportunistic fungal infections.

Complement C3 plays an essential role in the control of opportunistic fungal infections.

http://www.wikidata.org/entity/Q37333509

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New insights into innate immune control of systemic candidiasis Host response to Candida albicans bloodstream infection and sepsis Factor H Binds to Extracellular DNA Traps Released from Human Blood Monocytes in Response to Candida albicans.Influence of mannan and glucan on complement activation and C3 binding by Candida albicans.Candida glabrata persistence in mice does not depend on host immunosuppression and is unaffected by fungal amino acid auxotrophy.Genetic control of susceptibility to infection with Candida albicans in mice.Characterisation of innate fungal recognition in the lung Differential dependencies of monocytes and neutrophils on dectin-1, dectin-2 and complement for the recognition of fungal particles in inflammation Restoration of pattern recognition receptor costimulation to treat chromoblastomycosis, a chronic fungal infection of the skin Relative contributions of dectin-1 and complement to immune responses to particulate β-glucans.The contribution of mouse models to our understanding of systemic candidiasis.Immune defence against Candida fungal infections.Antifungal Immunological Defenses in Newborns.Analysis of the early cellular and humoral responses of Galleria mellonella larvae to infection by Candida albicans.Hypocomplementemia in kidney transplant recipients: impact on the risk of infectious complications.Candida glabrata intra-abdominal candidiasis is characterized by persistence within the peritoneal cavity and abscesses.Fungal Strategies to Evade the Host Immune Recognition.A Host-Pathogen Interaction Screen Identifies ada2 as a Mediator of Candida glabrata Defences Against Reactive Oxygen Species.Complement inhibition ameliorates blast-induced acute lung injury in rats: Potential role of complement in intracellular HMGB1-mediated inflammation Candida Immunity

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Complement C3 plays an essential role in the control of opportunistic fungal infections.

http://www.wikidata.org/entity/Q37333509

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 July 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Complement C3 plays an essential role in the control of opportunistic fungal infections.

@en

Complement C3 plays an essential role in the control of opportunistic fungal infections.

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type

label

Complement C3 plays an essential role in the control of opportunistic fungal infections.

@en

Complement C3 plays an essential role in the control of opportunistic fungal infections.

@nl

prefLabel

Complement C3 plays an essential role in the control of opportunistic fungal infections.

@en

Complement C3 plays an essential role in the control of opportunistic fungal infections.

@nl

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Infection and Immunity

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Complement C3 plays an essential role in the control of opportunistic fungal infections.

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Ann M Kerrigan

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Chad Steele

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Gordon D Brown

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Marina Botto

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Maureen Duffield

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Naren Srinivasan

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S Vicky Tsoni

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P2860

The beta-glucan receptor, dectin-1, is predominantly expressed on the surface of cells of the monocyte/macrophage and neutrophil lineages

Molecular organization and function of the complement system

Dectin-1 mediates the biological effects of beta-glucans

Recognition of microorganisms and activation of the immune response

Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity.

Comparison of pathogenesis and host immune responses to Candida glabrata and Candida albicans in systemically infected immunocompetent mice.

Pathogenicity of Saccharomyces cerevisiae in complement factor five-deficient mice.

An integrated model of the recognition of Candida albicans by the innate immune system.

A Saccharomyces cerevisiae mutant with increased virulence.

Distinct characteristics of initiation of the classical and alternative complement pathways by Candida albicans.

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3679-3685

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scholarly article

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10.1128/IAI.00233-09

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9

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77

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Philip R Taylor

Mohlopheni Marakalala

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2009-07-06T00:00:00Z

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19581397

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2738051

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