The nuclear-cytoplasmic shuttling of virion host shutoff RNase is enabled by pUL47 and an embedded nuclear export signal and defines the sites of degradation of AU-rich and stable cellular mRNAs
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Shutoff of Host Gene Expression in Influenza A Virus and Herpesviruses: Similar Mechanisms and Common ThemesTristetraprolin Recruits the Herpes Simplex Virion Host Shutoff RNase to AU-Rich Elements in Stress Response mRNAs To Enable Their Cleavage.Role of activating transcription factor 3 in the synthesis of latency-associated transcript and maintenance of herpes simplex virus 1 in latent state in ganglia.Emerging roles for RNA degradation in viral replication and antiviral defense.The 3 facets of regulation of herpes simplex virus gene expression: A critical inquiry.Molecular characterization of duck enteritis virus UL41 protein.
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The nuclear-cytoplasmic shuttling of virion host shutoff RNase is enabled by pUL47 and an embedded nuclear export signal and defines the sites of degradation of AU-rich and stable cellular mRNAs
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 09 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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The nuclear-cytoplasmic shuttl ...... rich and stable cellular mRNAs
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The nuclear-cytoplasmic shuttl ...... ich and stable cellular mRNAs.
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type
label
The nuclear-cytoplasmic shuttl ...... rich and stable cellular mRNAs
@en
The nuclear-cytoplasmic shuttl ...... ich and stable cellular mRNAs.
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prefLabel
The nuclear-cytoplasmic shuttl ...... rich and stable cellular mRNAs
@en
The nuclear-cytoplasmic shuttl ...... ich and stable cellular mRNAs.
@nl
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The nuclear-cytoplasmic shuttl ...... rich and stable cellular mRNAs
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Minfeng Shu
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P304
13569-13578
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10.1128/JVI.02603-13
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P577
2013-10-09T00:00:00Z