Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks) - Wikidata - wikimedia - TriplyDB

Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)

Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)

http://www.wikidata.org/entity/Q37340622

about

The KCNQ1 channel - remarkable flexibility in gating allows for functional versatility Adverse remodeling of the electrophysiological response to ischemia-reperfusion in human heart failure is associated with remodeling of metabolic gene expression.Arrhythmogenic cardiomyopathy in a patient with a rare loss-of-function KCNQ1 mutation.A long QT mutation substitutes cholesterol for phosphatidylinositol-4,5-bisphosphate in KCNQ1 channel regulation.Factors influencing success of clinical genome sequencing across a broad spectrum of disorders PIP2 regulation of KCNQ channels: biophysical and molecular mechanisms for lipid modulation of voltage-dependent gating.Voltage-Dependent Gating: Novel Insights from KCNQ1 Channels.Molecular Basis of Cardiac Delayed Rectifier Potassium Channel Function and Pharmacology.Potassium channels in the heart: structure, function and regulation.Inactivation of KCNQ1 potassium channels reveals dynamic coupling between voltage sensing and pore opening.Cryo-EM Structure of a KCNQ1/CaM Complex Reveals Insights into Congenital Long QT Syndrome.

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Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)

http://www.wikidata.org/entity/Q37340622

description

2013 nî lūn-bûn

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2013年の論文

@ja

2013年学术文章

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2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

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2013年學術文章

@zh-hant

name

Intracellular ATP binding is required to activate the slowly activating K+ channel I

@nl

Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)

@en

type

label

Intracellular ATP binding is required to activate the slowly activating K+ channel I

@nl

Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)

@en

prefLabel

Intracellular ATP binding is required to activate the slowly activating K+ channel I

@nl

Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)

@en

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PNAS.1315649110

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Intracellular ATP binding is required to activate the slowly activating K+ channel I(Ks)

@en

P2093

Ira S Cohen

http://www.w3.org/2001/XMLSchema#string

Jianmin Cui

http://www.w3.org/2001/XMLSchema#string

Jingyi Shi

http://www.w3.org/2001/XMLSchema#string

Junyuan Gao

http://www.w3.org/2001/XMLSchema#string

Kelli McFarland

http://www.w3.org/2001/XMLSchema#string

Kevin Bock

http://www.w3.org/2001/XMLSchema#string

Yang Li

http://www.w3.org/2001/XMLSchema#string

Zhongju Lu

http://www.w3.org/2001/XMLSchema#string

P2860

Requirement of a macromolecular signaling complex for beta adrenergic receptor modulation of the KCNQ1-KCNE1 potassium channel

Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel

Distantly related sequences in the alpha- and beta-subunits of ATP synthase, myosin, kinases and other ATP-requiring enzymes and a common nucleotide binding fold

Spectrum and prevalence of mutations from the first 2,500 consecutive unrelated patients referred for the FAMILION long QT syndrome genetic test

Molecular mechanism of ATP binding and ion channel activation in P2X receptors

A view of sur/KIR6.X, KATP channels

K(V)LQT1 and lsK (minK) proteins associate to form the I(Ks) cardiac potassium current

KCNE1 alters the voltage sensor movements necessary to open the KCNQ1 channel gate

KCNQ1 mutations in patients with a family history of lethal cardiac arrhythmias and sudden death.

Clinical practice. Long-QT syndrome.

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18922-18927

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scholarly article

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10.1073/PNAS.1315649110

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47

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110

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2013-11-04T00:00:00Z

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258282648

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24190995

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3839694

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