CXCR2 knockout mice are protected against DSS-colitis-induced acute kidney injury and inflammation.
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MyD88 mediates the protective effects of probiotics against the arteriolar thrombosis and leukocyte recruitment associated with experimental colitis.Giardia duodenalis infection reduces granulocyte infiltration in an in vivo model of bacterial toxin-induced colitis and attenuates inflammation in human intestinal tissueOligomannan Prebiotic Attenuates Immunological, Clinical and Behavioral Symptoms in Mouse Model of Inflammatory Bowel Disease.SIRT2 Regulates LPS-Induced Renal Tubular CXCL2 and CCL2 Expression.A20 regulates IL-1-induced tolerant production of CXC chemokines in human mesangial cells via inhibition of MAPK signalingNeutropenic sepsis is associated with distinct clinical and biological characteristics: a cohort study of severe sepsisAutophagy deficiency in myeloid cells increases susceptibility to obesity-induced diabetes and experimental colitis.Transglutaminase 2 is dispensable but required for the survival of mice in dextran sulfate sodium-induced colitisCXCR2 deficient mice display macrophage-dependent exaggerated acute inflammatory responses.Neutrophils in ulcerative colitis: a review of selected biomarkers and their potential therapeutic implications.Colitis ImmunoPET: Defining Target Cell Populations and Optimizing Pharmacokinetics.Evaluating [11C]PBR28 PET for Monitoring Gut and Brain Inflammation in a Rat Model of Chemically Induced Colitis.Dextran sodium sulfate (DSS) induces necrotizing enterocolitis-like lesions in neonatal miceRegulation of Chemokine Function: The Roles of GAG-Binding and Post-Translational NitrationFunctional characterization of CXCR4 in mediating the expression of protein C system in experimental ulcerative colitis.Guar gum fiber increases suppressor of cytokine signaling-1 expression via toll-like receptor 2 and dectin-1 pathways, regulating inflammatory response in small intestinal epithelial cells.The enhanced susceptibility of ADAM-17 hypomorphic mice to DSS-induced colitis is not ameliorated by loss of RIPK3, revealing an unexpected function of ADAM-17 in necroptosis.The enhanced susceptibility of ADAM-17 hypomorphic mice to DSS-induced colitis is not ameliorated by loss of RIPK3, revealing an unexpected function of ADAM-17 in necroptosis.Pharmacological activation of epidermal growth factor receptor signaling inhibits colitis-associated cancer in mice.The Gut-Kidney Axis: Putative Interconnections Between Gastrointestinal and Renal Disorders
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CXCR2 knockout mice are protected against DSS-colitis-induced acute kidney injury and inflammation.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 August 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
CXCR2 knockout mice are protec ...... idney injury and inflammation.
@en
CXCR2 knockout mice are protec ...... idney injury and inflammation.
@nl
type
label
CXCR2 knockout mice are protec ...... idney injury and inflammation.
@en
CXCR2 knockout mice are protec ...... idney injury and inflammation.
@nl
prefLabel
CXCR2 knockout mice are protec ...... idney injury and inflammation.
@en
CXCR2 knockout mice are protec ...... idney injury and inflammation.
@nl
P2093
P2860
P1476
CXCR2 knockout mice are protec ...... idney injury and inflammation.
@en
P2093
Calpurnia Jayakumar
Ganesan Ramesh
Punithavathi Ranganathan
Santhakumar Manicassamy
P2860
P304
P356
10.1152/AJPRENAL.00319.2013
P577
2013-08-28T00:00:00Z