Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility. - Wikidata - wikimedia - TriplyDB

Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility.

Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility.

http://www.wikidata.org/entity/Q37342498

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From integrative genomics to systems genetics in the rat to link genotypes to phenotypes Polyamines regulate the stability of JunD mRNA by modulating the competitive binding of its 3' untranslated region to HuR and AUF1 Naturally occurring genetic variability in expression of Gsta4 is associated with differential survival of axotomized rat motoneurons Macrophage epoxygenase determines a profibrotic transcriptome signature.Identification of a nutrient-sensing transcriptional network in monocytes by using inbred rat models on a cafeteria diet.Alternative splicing and transcriptome profiling of experimental autoimmune encephalomyelitis using genome-wide exon arrays.Non-thermal plasma activates human keratinocytes by stimulation of antioxidant and phase II pathways.Mannose receptor interacts with Fc receptors and is critical for the development of crescentic glomerulonephritis in mice.Combined protein- and nucleic acid-level effects of rs1143679 (R77H), a lupus-predisposing variant within ITGAM Identification of Ceruloplasmin as a Gene that Affects Susceptibility to Glomerulonephritis Through Macrophage Function.BCAT1 controls metabolic reprogramming in activated human macrophages and is associated with inflammatory diseases Macrophages and immunologic inflammation of the kidney.Pigment epithelium-derived factor is an intrinsic antifibrosis factor targeting hepatic stellate cells Identification of a novel gene for diabetic traits in rats, mice, and humans.Genetics of hypertension: from experimental animals to humans.Comparison of small interfering RNA (siRNA) delivery into bovine monocyte-derived macrophages by transfection and electroporation.Genetic susceptibility and loss of Nr4a1 enhances macrophage-mediated renal injury in CKD.Combined ChIP-Seq and transcriptome analysis identifies AP-1/JunD as a primary regulator of oxidative stress and IL-1β synthesis in macrophages.JunD/AP1 regulatory network analysis during macrophage activation in a rat model of crescentic glomerulonephritis.AP-1 transcription factor JunD confers protection from accelerated nephrotoxic nephritis and control podocyte-specific Vegfa expression.JunD enhances miR-29b levels transcriptionally and posttranscriptionally to inhibit proliferation of intestinal epithelial cells.Autoimmunity to the alpha 3 chain of type IV collagen in glomerulonephritis is triggered by 'autoantigen complementarity'.Kcnn4 is a regulator of macrophage multinucleation in bone homeostasis and inflammatory disease Role of novel rat-specific Fc receptor in macrophage activation associated with crescentic glomerulonephritis A Bayesian Approach for Analysis of Whole-Genome Bisulfite Sequencing Data Identifies Disease-Associated Changes in DNA Methylation Genetic susceptibility to experimental autoimmune glomerulonephritis in the Wistar Kyoto rat.New Wistar Kyoto and spontaneously hypertensive rat transgenic models with ubiquitous expression of green fluorescent protein Experimental crescentic glomerulonephritis: a new bicongenic rat model.The role of stress-activated protein kinase signaling in renal pathophysiology.Combining genetic mapping with genome-wide expression in experimental autoimmune encephalomyelitis highlights a gene network enriched for T cell functions and candidate genes regulating autoimmunity.The molecular pathogenesis of HIV-1 associated nephropathy: recent advances.Strain differences and the genetic basis of experimental autoimmune anti-glomerular basement membrane glomerulonephritis.The AP-1 Transcription Factor c-Jun Promotes Arthritis by Regulating Cyclooxygenase-2 and Arginase-1 Expression in Macrophages.Transcriptional regulation of importin-α1 by JunD modulates subcellular localization of RNA-binding protein HuR in intestinal epithelial cells.A susceptibility gene for kidney disease in an obese mouse model of type II diabetes maps to chromosome 8.Differential 12-O-Tetradecanoylphorbol-13-acetate-induced activation of rat mammary carcinoma susceptibility Fbxo10 variant promoters via a PKC-AP1 pathway.Characterization of the macrophage transcriptome in glomerulonephritis-susceptible and -resistant rat strains.IL-10 inhibits transcription elongation of the human TNF gene in primary macrophages.Unique regulatory properties of mesangial cells are genetically determined in the rat.Automatic identification of informative regions with epigenomic changes associated to hematopoiesis.

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P2860

Jund is a determinant of macrophage activation and is associated with glomerulonephritis susceptibility.

http://www.wikidata.org/entity/Q37342498

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article científic

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scientific article published on May 2008

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name

Jund is a determinant of macro ...... erulonephritis susceptibility.

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Jund is a determinant of macro ...... erulonephritis susceptibility.

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type

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Jund is a determinant of macro ...... erulonephritis susceptibility.

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Jund is a determinant of macro ...... erulonephritis susceptibility.

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prefLabel

Jund is a determinant of macro ...... erulonephritis susceptibility.

@en

Jund is a determinant of macro ...... erulonephritis susceptibility.

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Nature Genetics

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Jund is a determinant of macro ...... erulonephritis susceptibility.

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Alan Salama

http://www.w3.org/2001/XMLSchema#string

Brenda Mutch

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Brian M Foxwell

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Charles D Pusey

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Gurjeet Bhangal

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H Terence Cook

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Jacques Behmoaras

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Jan Domin

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Jennifer Smith

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Kylie McDonald

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P2860

Transgenic rescue of defective Cd36 ameliorates insulin resistance in spontaneously hypertensive rats

Involvement of the Arp2/3 complex in phagocytosis mediated by FcgammaR or CR3.

Copy number polymorphism in Fcgr3 predisposes to glomerulonephritis in rats and humans.

Conditional ablation of macrophages halts progression of crescentic glomerulonephritis.

Evidence for a pathogenic role of a cell-mediated immune mechanism in experimental glomerulonephritis.

JunD regulates lymphocyte proliferation and T helper cell cytokine expression

JunD protects cells from p53-dependent senescence and apoptosis.

Functional characteristics of macrophages in glomerulonephritis in the rat. O2- generation, MHC class II expression, and eicosanoid synthesis.

Mouse JunD negatively regulates fibroblast growth and antagonizes transformation by ras.

Signal transducer and activator of transcription 3 is the dominant mediator of the anti-inflammatory effects of IL-10 in human macrophages.

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553-559

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scholarly article

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10.1038/NG.137

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5

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40

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2008-05-01T00:00:00Z

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5408002

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18443593

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glomerulonephritis

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2742200

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