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Termination of NF-kappaB activity through a gammaherpesvirus protein that assembles an EC5S ubiquitin-ligaseImmune interventions in strokeImmune mechanisms in cerebral ischemic toleranceThe Myelin and Lymphocyte Protein MAL Is Required for Binding and Activity of Clostridium perfringens ε-ToxinAngiotensin II impairs neurovascular coupling in neocortex through NADPH oxidase-derived radicalsKey role of tissue plasminogen activator in neurovascular couplingNox2-derived radicals contribute to neurovascular and behavioral dysfunction in mice overexpressing the amyloid precursor protein.Angiotensin II attenuates functional hyperemia in the mouse somatosensory cortex.Abeta-induced vascular oxidative stress and attenuation of functional hyperemia in mouse somatosensory cortex.Exogenous NADPH increases cerebral blood flow through NADPH oxidase-dependent and -independent mechanisms.Angiotensin II attenuates endothelium-dependent responses in the cerebral microcirculation through nox-2-derived radicals.Cerebrovascular nitrosative stress mediates neurovascular and endothelial dysfunction induced by angiotensin II.Nox2-derived reactive oxygen species mediate neurovascular dysregulation in the aging mouse brain.Estrous cycle-dependent neurovascular dysfunction induced by angiotensin II in the mouse neocortex.Cyclooxygenase 1-derived prostaglandin E2 and EP1 receptors are required for the cerebrovascular dysfunction induced by angiotensin II.Size-selective opening of the blood-brain barrier by targeting endothelial sphingosine 1-phosphate receptor 1.Purinergic signaling induces cyclooxygenase-1-dependent prostanoid synthesis in microglia: roles in the outcome of excitotoxic brain injury.Key role of CD36 in Toll-like receptor 2 signaling in cerebral ischemia.Chronic intermittent hypoxia induces NMDA receptor-dependent plasticity and suppresses nitric oxide signaling in the mouse hypothalamic paraventricular nucleus.Stroke research at a crossroad: asking the brain for directionsMonoubiquitination of nuclear RelA negatively regulates NF-κB activity independent of proteasomal degradationEndothelin 1-dependent neurovascular dysfunction in chronic intermittent hypoxia.cis-acting, element-specific transcriptional activity of differentially phosphorylated nuclear factor-kappa B.Regulation of nuclear factor κB (NF-κB) transcriptional activity via p65 acetylation by the chaperonin containing TCP1 (CCT)Scavenger receptor CD36 is essential for the cerebrovascular oxidative stress and neurovascular dysfunction induced by amyloid-beta.The role of microglia and myeloid immune cells in acute cerebral ischemia.SUMO2/3 is associated with ubiquitinated protein aggregates in the mouse neocortex after middle cerebral artery occlusionAngiotensin II-dependent hypertension requires cyclooxygenase 1-derived prostaglandin E2 and EP1 receptor signaling in the subfornical organ of the brain.Central cardiovascular circuits contribute to the neurovascular dysfunction in angiotensin II hypertension.Angiotensin II type 2 receptor-coupled nitric oxide production modulates free radical availability and voltage-gated Ca2+ currents in NTS neurons.Reperfusion rather than ischemia drives the formation of ubiquitin aggregates after middle cerebral artery occlusionEndothelial CD36 Contributes to Postischemic Brain Injury by Promoting Neutrophil Activation via CSF3.Hypo-phosphorylation leads to nuclear retention of NF-kappaB p65 due to impaired IkappaBalpha gene synthesis.Site-specific phosphorylation of the p65 protein subunit mediates selective gene expression by differential NF-κB and RNA polymerase II promoter recruitmentHypertension enhances Aβ-induced neurovascular dysfunction, promotes β-secretase activity, and leads to amyloidogenic processing of APPInnate immunity receptor CD36 promotes cerebral amyloid angiopathyThe ubiquitin ligase HERC3 attenuates NF-κB-dependent transcription independently of its enzymatic activity by delivering the RelA subunit for degradationCommensal microbiota affects ischemic stroke outcome by regulating intestinal γδ T cells.Angiotensin II slow-pressor hypertension enhances NMDA currents and NOX2-dependent superoxide production in hypothalamic paraventricular neurons.Nuclear factor-kappaB activation and postischemic inflammation are suppressed in CD36-null mice after middle cerebral artery occlusion.
P50
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P50
description
onderzoeker
@nl
name
Josef Anrather
@ast
Josef Anrather
@en
Josef Anrather
@es
Josef Anrather
@nl
Josef Anrather
@sl
type
label
Josef Anrather
@ast
Josef Anrather
@en
Josef Anrather
@es
Josef Anrather
@nl
Josef Anrather
@sl
prefLabel
Josef Anrather
@ast
Josef Anrather
@en
Josef Anrather
@es
Josef Anrather
@nl
Josef Anrather
@sl