How do kidney cells adapt to survive in hypertonic inner medulla? - Wikidata - wikimedia - TriplyDB

How do kidney cells adapt to survive in hypertonic inner medulla?

How do kidney cells adapt to survive in hypertonic inner medulla?

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Organic osmolytes preserve the function of the developing tight junction in ultraviolet B-irradiated rat epidermal keratinocytes.More complete polarization of renal tubular epithelial cells by artificial urine

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How do kidney cells adapt to survive in hypertonic inner medulla?

http://www.wikidata.org/entity/Q37348143

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on January 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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How do kidney cells adapt to survive in hypertonic inner medulla?

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How do kidney cells adapt to survive in hypertonic inner medulla?

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How do kidney cells adapt to survive in hypertonic inner medulla?

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How do kidney cells adapt to survive in hypertonic inner medulla?

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How do kidney cells adapt to survive in hypertonic inner medulla?

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How do kidney cells adapt to survive in hypertonic inner medulla?

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Transactions of the American Clinical and Climatological Association

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How do kidney cells adapt to survive in hypertonic inner medulla?

@en

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Tomas Berl

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Tonicity-responsive enhancer binding protein, a rel-like protein that stimulates transcription in response to hypertonicity

Multi-PDZ domain protein 1 (MUPP1) is concentrated at tight junctions through its possible interaction with claudin-1 and junctional adhesion molecule

Differential expression patterns of claudins, tight junction membrane proteins, in mouse nephron segments

Distribution and oligomeric association of splice forms of Na(+)-K(+)-ATPase regulatory gamma-subunit in rat kidney

Claudins--key pieces in the tight junction puzzle.

The gamma subunit modulates Na(+) and K(+) affinity of the renal Na,K-ATPase.

The expression of the gamma subunit of Na-K-ATPase is regulated by osmolality via C-terminal Jun kinase and phosphatidylinositol 3-kinase-dependent mechanisms.

TonEBP transcriptional activator in the cellular response to increased osmolality.

Cell-specific expression of three members of the FXYD family along the renal tubule.

Chloride, not sodium, stimulates expression of the gamma subunit of Na/K-ATPase and activates JNK in response to hypertonicity in mouse IMCD3 cells

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389-401

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scholarly article

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120

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2009-01-01T00:00:00Z

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19768191

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2744507

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