Cell cycle-dependent phosphorylation of C/EBPbeta mediates oncogenic cooperativity between C/EBPbeta and H-RasV12.
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Functional analysis of novel phosphorylation sites of CREB-binding protein using mass spectrometry and mammalian two-hybrid assaysUp-regulation of c-Jun-NH2-kinase pathway contributes to the induction of mitochondria-mediated apoptosis by alpha-tocopheryl succinate in human prostate cancer cells.The importance of being flexible: the case of basic region leucine zipper transcriptional regulators.Epidermal growth factor-induced GnRH-II synthesis contributes to ovarian cancer cell invasionNSAID-activated gene-1 as a molecular target for capsaicin-induced apoptosis through a novel molecular mechanism involving GSK3beta, C/EBPbeta and ATF3.RSK-mediated phosphorylation in the C/EBP{beta} leucine zipper regulates DNA binding, dimerization, and growth arrest activityCCAAT/Enhancer-binding protein beta DNA binding is auto-inhibited by multiple elements that also mediate association with p300/CREB-binding protein (CBP).C/EBPbeta cooperates with RB:E2F to implement Ras(V12)-induced cellular senescence.Negative regulation of C/EBPbeta1 by sumoylation in breast cancer cells.The transcription factor C/EBP-β mediates constitutive and LPS-inducible transcription of murine SerpinB2.CCAAT/enhancer-binding protein beta: its role in breast cancer and associations with receptor tyrosine kinases.AML1/RUNX1 phosphorylation by cyclin-dependent kinases regulates the degradation of AML1/RUNX1 by the anaphase-promoting complex3'UTR elements inhibit Ras-induced C/EBPβ post-translational activation and senescence in tumour cellsG9a-mediated lysine methylation alters the function of CCAAT/enhancer-binding protein-beta.Transcription profiling of C/EBP targets identifies Per2 as a gene implicated in myeloid leukemiaRole of cdk2 in the sequential phosphorylation/activation of C/EBPbeta during adipocyte differentiationOncogenic KRAS impairs EGFR antibodies' efficiency by C/EBPβ-dependent suppression of EGFR expressionTRB3 blocks adipocyte differentiation through the inhibition of C/EBPbeta transcriptional activityHistone demethylase Kdm4b functions as a co-factor of C/EBPβ to promote mitotic clonal expansion during differentiation of 3T3-L1 preadipocytes.The interferon signaling network and transcription factor C/EBP-betaConditional ablation of C/EBP beta demonstrates its keratinocyte-specific requirement for cell survival and mouse skin tumorigenesis.C/EBPbeta represses p53 to promote cell survival downstream of DNA damage independent of oncogenic Ras and p19(Arf).Ha-Ras transformation of MCF10A cells leads to repression of Singleminded-2s through NOTCH and C/EBPbeta.RasV12-mediated down-regulation of CCAAT/enhancer binding protein beta in immortalized fibroblasts requires loss of p19Arf and facilitates bypass of oncogene-induced senescenceC/EBPalpha expression is partially regulated by C/EBPbeta in response to DNA damage and C/EBPalpha-deficient fibroblasts display an impaired G1 checkpointF-BOX proteins in cancer cachexia and muscle wasting: Emerging regulators and therapeutic opportunities.Constitutively active RAS signaling reduces 1,25 dihydroxyvitamin D-mediated gene transcription in intestinal epithelial cells by reducing vitamin D receptor expression.Interaction and cooperation of the CCAAT-box enhancer-binding protein β (C/EBPβ) with the homeodomain-interacting protein kinase 2 (Hipk2).Regulation of C/EBPbeta1 by Ras in mammary epithelial cells and the role of C/EBPbeta1 in oncogene-induced senescence.Fibronectin upregulates cGMP-dependent protein kinase type Iβ through C/EBP transcription factor activation in contractile cells.Cyclic GMP-dependent protein kinase regulates CCAAT enhancer-binding protein beta functions through inhibition of glycogen synthase kinase-3.A role for mixed lineage kinases in regulating transcription factor CCAAT/enhancer-binding protein-{beta}-dependent gene expression in response to interferon-{gamma}.Cell cycle defects contribute to a block in hormone-induced mammary gland proliferation in CCAAT/enhancer-binding protein (C/EBPbeta)-null mice.
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Cell cycle-dependent phosphorylation of C/EBPbeta mediates oncogenic cooperativity between C/EBPbeta and H-RasV12.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 2004
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Cell cycle-dependent phosphory ...... etween C/EBPbeta and H-RasV12.
@en
Cell cycle-dependent phosphory ...... etween C/EBPbeta and H-RasV12.
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type
label
Cell cycle-dependent phosphory ...... etween C/EBPbeta and H-RasV12.
@en
Cell cycle-dependent phosphory ...... etween C/EBPbeta and H-RasV12.
@nl
prefLabel
Cell cycle-dependent phosphory ...... etween C/EBPbeta and H-RasV12.
@en
Cell cycle-dependent phosphory ...... etween C/EBPbeta and H-RasV12.
@nl
P2093
P2860
P50
P1476
Cell cycle-dependent phosphory ...... between C/EBPbeta and H-RasV12
@en
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Robert C Smart
Songyun Zhu
Terry D Copeland
Thomas Sebastian
P2860
P304
P356
10.1128/MCB.24.17.7380-7391.2004
P407
P577
2004-09-01T00:00:00Z