Pyloric sphincter dysfunction in nNOS-/- and W/Wv mutant mice: animal models of gastroparesis and duodenogastric reflux - Wikidata - wikimedia - TriplyDB

Pyloric sphincter dysfunction in nNOS-/- and W/Wv mutant mice: animal models of gastroparesis and duodenogastric reflux

Pyloric sphincter dysfunction in nNOS-/- and W/Wv mutant mice: animal models of gastroparesis and duodenogastric reflux

http://www.wikidata.org/entity/Q37349922

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Mounting evidence against the role of ICC in neurotransmission to smooth muscle in the gut.Neurochemical phenotypes of myenteric neurons in the rhesus monkey.Cellular pathogenesis of diabetic gastroenteropathy.The significance of interstitial cells in neurogastroenterology Interstitial cells: regulators of smooth muscle function.Neuroeffector apparatus in gastrointestinal smooth muscle organs.Intraesophageal manganese superoxide dismutase-plasmid liposomes ameliorates novel total-body and thoracic radiation sensitivity of NOS1-/- mice.Pathophysiology of colonic causes of chronic constipation Review article: gastric electrical stimulation for gastroparesis--physiological foundations, technical aspects and clinical implications.Use of fluoroscopy to study in vivo motility in mouse pups.Common Mechanism of Pathogenesis in Gastrointestinal Diseases Implied by Consistent Efficacy of Single Chinese Medicine Formula: A PRISMA-Compliant Systematic Review and Meta-Analysis Is Interstitial Cells of Cajal‒opathy Present in Gastroparesis?Reduction of gastrointestinal motility by unilateral thyroparathyroidectomy plus subdiaphragmatic vagotomy in rats.Recent advances in the pathophysiology and treatment of gastroparesis.Bioengineered Human Pyloric Sphincters Using Autologous Smooth Muscle and Neural Progenitor Cells.Inflammation induced by mast cell deficiency rather than the loss of interstitial cells of Cajal causes smooth muscle dysfunction in W/W(v) mice.Electroacupuncture at ST36 Protects ICC Networks via mSCF/Kit-ETV1 Signaling in the Stomach of Diabetic Mice.Infantile hypertrophic pyloric stenosis--genetics and syndromes.Toward a better understanding of gastrointestinal nitrergic neuromuscular transmission.Convergence of inhibitory neural inputs regulate motor activity in the murine and monkey stomach.Delayed gastric emptying and enteric nervous system dysfunction in the rotenone model of Parkinson's disease.Interstitial cells of Cajal integrate excitatory and inhibitory neurotransmission with intestinal slow-wave activity.Tetrahydrobiopterin deficiency induces gastroparesis in newborn mice.Infantile hypertrophic pyloric stenosis (IHPS): a study of its pathophysiology utilizing the newborn hph-1 mouse model of the disease.Effect of Hyperglycemia on Purinergic and Nitrergic Inhibitory Neuromuscular Transmission in the Antrum of the Stomach: Implications for Fast Gastric Emptying.Gender and estradiol as major factors in the expression and dimerization of nNOSα in rats with experimental diabetic gastroparesis.Class side effects: decreased pressure in the lower oesophageal and the pyloric sphincters after the administration of dopamine antagonists, neuroleptics, anti-emetics, L-NAME, pentadecapeptide BPC 157 and L-arginine.CrossTalk opposing view: Interstitial cells are not involved and physiologically important in neuromuscular transmission in the gut.

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Pyloric sphincter dysfunction in nNOS-/- and W/Wv mutant mice: animal models of gastroparesis and duodenogastric reflux

http://www.wikidata.org/entity/Q37349922

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article científic

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article scientifique

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articolo scientifico

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bilimsel makale

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scientific article published on 20 June 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Pyloric sphincter dysfunction ...... esis and duodenogastric reflux

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Pyloric sphincter dysfunction ...... sis and duodenogastric reflux.

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Pyloric sphincter dysfunction ...... esis and duodenogastric reflux

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Pyloric sphincter dysfunction ...... sis and duodenogastric reflux.

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Pyloric sphincter dysfunction ...... esis and duodenogastric reflux

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Pyloric sphincter dysfunction ...... sis and duodenogastric reflux.

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J.GASTRO.2008.06.039

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Gastroenterology

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Pyloric sphincter dysfunction ...... esis and duodenogastric reflux

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Digavalli V Sivarao

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Hiroshi Mashimo

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Raj K Goyal

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P2860

Selective knockout of intramuscular interstitial cells reveals their role in the generation of slow waves in mouse stomach

Disturbed pyloric motility in Ws/Ws mutant rats due to deficiency of c-kit-expressing interstitial cells of Cajal.

Disturbed intestinal movement, bile reflux to the stomach, and deficiency of c-kit-expressing cells in Ws/Ws mutant rats.

Regulation of interstitial cells of Cajal in the mouse gastric body by neuronal nitric oxide.

Physiology and pathophysiology of the interstitial cell of Cajal: from bench to bedside. II. Gastric motility: lessons from mutant mice on slow waves and innervation.

Physiology and pathophysiology of the interstitial cells of Cajal: from bench to bedside. IV. Genetic and animal models of GI motility disorders caused by loss of interstitial cells of Cajal.

Extrinsic and intrinsic neural control of pyloric sphincter pressure in the dog.

Mechanisms of disease: the pathological basis of gastroparesis--a review of experimental and clinical studies.

Gastroduodenal motility disturbances in man.

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1258-1266

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10.1053/J.GASTRO.2008.06.039

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